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- Biology of the Hair Follicle: The Basics
- Physical and Functional Interaction between the Vitamin D Receptor and Hairless Corepressor, Two Proteins Required for Hair Cycling
- Loss-of-function mutations of an inhibitory upstream ORF in the human hairless transcript cause Marie Unna hereditary hypotrichosis
- The <i>hairless</i> gene mutated in congenital hair loss disorders encodes a novel nuclear receptor corepressor
- The Role of the Hairless (hr) Gene in the. Regulation of Hair Follicle Catagen Transformation
- Prostaglandin-Induced Hair Growth
- Atrichia Caused by Mutations in the Vitamin D Receptor Gene is a Phenocopy of Generalized Atrichia Caused by Mutations in the Hairless Gene
- Hairless triggers reactivation of hair growth by promoting Wnt signaling
- Molecular and functional aspects of the hairless (<i>hr</i>) gene in laboratory rodents and humans
- Towards Defining the Pathogenesis of the Hairless Phenotype
- A Missense Mutation in the Zinc-Finger Domain of the Human Hairless Gene Underlies Congenital Atrichia in a Family of Irish Travellers
- Hairless Suppresses Vitamin D Receptor Transactivation in Human Keratinocytes
- Hairless and Wnt Signaling: Allies in Epithelial Stem Cell Differentiation
- Therapeutic strategy for hair regeneration: hair cycle activation, niche environment modulation, wound-induced follicle neogenesis, and stem cell engineering
- Novel Hairless Mutations in Two Kindreds with Autosomal Recessive Papular Atrichia
- Relation of Skin Polyamines to the Hairless Phenotype in Transgenic Mice Overexpressing Spermidine/Spermine N1-Acetyltransferase
- Characterization of hairless (Hr) and FGF5genes provides insights into the molecular basis of hair loss in cetaceans
- A novel missense mutation (C622G) in the zinc‐finger domain of the human hairless gene associated with congenital atrichia with papular lesions
- Interactions of the Vitamin D Receptor with the Corepressor Hairless
- A Homozygous Nonsense Mutation in the Zinc-Finger Domain of the Human Hairless Gene Underlies Congenital Atrichia
- Evaluation of the Hairless Rat as a Model for in Vivo Percutaneous Absorption
- A distinct gene close to the hairless locus on chromosome 8p underlies hereditary Marie Unna type hypotrichosis in a German family
- The character “hairless” in the mouse
- Ion pairs of risedronate for transdermal delivery and enhanced permeation rate on hairless mouse skin
- Hairless is a nuclear receptor corepressor essential for skin function
- Hairless Modulates Ligand-Dependent Activation of the Vitamin D Receptor-Retinoid X Receptor Heterodimer
- Intragenic deletion in the Desmoglein 4 gene underlies the skin phenotype in the Iffa Credo “hairless” rat
- The mouse frizzy (<i>fr</i>) and rat ‘hairless’ (<i>fr</i><sup><i>CR</i></sup>) mutations are natural variants of protease serine S1 family member 8 (<i>Prss8</i>)
- Werewolf, There Wolf: Variants in Hairless Associated with Hypotrichia and Roaning in the Lykoi Cat Breed
- A Novel Missense Mutation Affecting the Human Hairless Thyroid Receptor Interacting Domain 2 Causes Congenital Atrichia