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The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

Chemotherapy and radiation therapy cause skin and hair damage by altering gene expression and signaling pathways.

The BMP/Smads pathway and Id2 gene control hair follicle stem cells, affecting their rest and growth phases.

Scientists found gene mutations that affect hair loss, skin stem cells, and skin disorders, and identified drugs that may help treat blood vessel and skin conditions.

The document concludes that Borrelia afzelii causes a skin condition in France, a gene is linked to hair loss in Caucasian women, and various genetic mutations affect skin diseases.

The document concludes that understanding hair biology is key to treating hair disorders, with gene therapy showing potential as a future treatment.

Hair pattern in androgenetic alopecia overlaps with scalp and bone demarcations, with distinct gene profiles affecting susceptibility.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Certain mutations in a hair growth-related gene cause a type of genetic hair loss.

Mutations in the Vitamin D receptor gene can cause hair loss similar to mutations in the Hairless gene.

Involucrin gene expression is controlled by specific proteins and signaling pathways.

GABRA2 gene variations impact alcohol response, and hair loss medication finasteride reduces some effects.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

EDA2R gene linked to hair loss.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

E211 G>A gene linked to lower risk of severe prostate cancer and hair loss.

A deficiency in the TTC7A gene causes immune problems, gut issues, and hair loss.

Mutations in the LSS gene cause a rare type of hereditary hair loss.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

CGRP may help protect hair follicles from immune system attacks, potentially slowing hair loss.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Mutations in the PTPRQ gene cause significant balance issues in mice due to hair bundle defects in the inner ear.

New therapies for Birt–Hogg–Dubé syndrome are being developed based on understanding the FLCN gene's role.

A gene in Arabidopsis thaliana, AtPRPL1, affects root hair length but not cell wall composition.

A specific protein complex increases the activity of a plant enzyme, but this action is not required for plant root hair growth.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

Certain HSD3B1 gene types are linked to worse prostate cancer outcomes and affect treatment response and other health conditions.

Human keratinocytes do not naturally respond to androgens.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.