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Gene network oscillations inside hair stem cells are key for hair growth regulation and could help treat hair loss.

Small molecule IM boosts hair growth by changing stem cell metabolism.

A gene network led by RSL4 is crucial for early root hair growth in response to cold in Arabidopsis thaliana.

Hairless protein can block vitamin D activation in skin cells.

The Ar/miR-221/IGF-1 pathway is involved in male pattern baldness, with miR-221 potentially being a new target for treatment.

Faulty LEF1 activation causes faster skin cell differentiation in premature aging syndrome.

Dlx3 is essential for hair growth and regeneration.

A specific pathway involving AR, miR-221, and IGF-1 plays a key role in causing common hair loss.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Skin cell types develop when specific genes are turned on by removing certain chemical tags from DNA.

Hair follicle stem cells use specific chromatin changes to control their growth and differentiation.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Stem cell plasticity is crucial for wound healing but can also contribute to cancer development.

Different signals work together to change gene activity and guide hair follicle stem cells to become specific cell types.

Brg1 is crucial for hair growth and skin repair by maintaining stem cells and promoting regeneration.

Short-chain fatty acids from *Cutibacterium acnes* cause skin inflammation, contributing to acne.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

MicroRNAs are crucial in controlling cell signaling, affecting cancer and tissue regeneration.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

The circadian clock affects skin stem cell behavior, impacting aging and cancer risk.

The timing of when the gene Bmal1 is active affects aging and survival, with its absence during development, not adulthood, leading to premature aging.

Damage to skin releases dsRNA, which activates TLR3 and helps in skin and hair follicle regeneration.

The AUTS2 gene is linked to neurological disorders and may affect human brain development and cognition.