Search

everythinglearnresearchcommunity

for

Search:↓

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

ATIR101 improves survival in stem cell transplant patients; Australian stem cell treatment decisions are influenced by regulation changes.

The calcineurin/NFAT pathway plays a significant role in the development and growth of a type of skin cancer called cutaneous squamous cell carcinoma.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

The study's results on the effectiveness of low-dose IL-2 for alopecia areata and its impact on immune cells were not provided.

Different markers are found in stem cells of the scalp's hair follicle bulge and the surrounding skin.

Alopecia areata is a hair loss condition caused by immune factors and can be treated with JAK inhibitors.

Patients with RASopathies have a higher risk of autoimmune disorders and should be routinely screened.

Recognizing the different stages of alopecia areata is crucial for accurate diagnosis and treatment.

Using fat-derived stem cells with the drug meglumine antimoniate can help control skin disease and reduce parasites in mice with leishmaniasis.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

Alopecia areata shows a unique type 1 interferon signature, suggesting potential treatment by targeting this pathway.

Men are more likely to have severe respiratory viral infections like COVID-19 due to hormonal and genetic differences, while women generally have stronger immune responses.

Hair regrowth was seen in 83% of children with alopecia, immune system plays a role in the condition, and various treatments showed effectiveness for hair and nail disorders.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

Alopecia areata may be treated by restoring hair follicle immune privilege and adjusting immune responses.

Different types of skin cells and immune cells play a role in healing UV-damaged skin, with chronic UV exposure causing lasting damage to certain skin cells.

Patients with RASopathies are at risk for autoimmune disorders and should be routinely screened.

Cells from the lower part of hair follicles are a promising, less invasive option for immune system therapies.

Unconventional lymphocytes are important for quick immune responses and healing of skin and mucosal barriers.

New insights into cell communication in psoriasis suggest innovative drug treatments.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Hair follicle stem cells are a promising source for tissue repair and treating skin or hair diseases.

Environmental cues can change the fate and function of epithelial cells, with potential for cell therapy.

Testosterone can both promote hair growth and cause baldness by affecting hair growth signals.

HDAC inhibitors, like Vorinostat and Entinostat, can help regrow hair in alopecia areata.

Dermal Papilla cells are a promising tool for evaluating hair growth treatments.

Lower CD200R1 on certain immune cells is linked to more severe rheumatoid arthritis and immune imbalance.

Folliculotropic mycosis fungoides has a worse prognosis than other types, with survival rates varying significantly based on subtype and organ involvement.