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Hair mineral analysis might help diagnose diseases early, but standard methods are needed.

The conclusion is that managing androgen excess requires long-term treatment, including hormonal contraceptives and androgen blockers, with follow-up after six months.

A three-month treatment with a GnRH agonist significantly lowered androgen levels and 5α-reductase activity in men with benign prostatic hyperplasia.

Women with PCOS have higher androgen levels that decrease with age but are still higher than in women without PCOS.

Many young women who donate blood have hormonal disorders like excess male hormones and PCOS.

Androgens slow hair growth by altering Wnt signaling in balding cells.

The document concludes that targeting the androgen receptor may be a promising breast cancer treatment, especially for certain types.

Androgens can both promote and prevent hair growth due to differences in gene expression in hair follicles.

Researchers found 63 genes linked to male-pattern baldness, which could help in understanding its biology and developing new treatments.

Androgens are important for normal ovarian function and estrogen production, but may not be the main cause of follicle death.

The balance between androgen receptor and p53 is crucial for sebaceous gland differentiation.

Androgens increase a growth factor in hair cells by creating reactive oxygen species, and antioxidants might help treat hair loss.

The length of the CAG repeat in the androgen receptor gene affects the risk and progression of prostate cancer, BPH, infertility, and undermasculinized genitalia.

Androgen conjugates might be better indicators of skin sensitivity to hormones in women with excessive hair growth.

Dietary changes, including weight loss and a balanced diet, are important for managing PCOS, especially in overweight women.

Low-level laser therapy may improve hair regrowth and thickness for androgenetic alopecia, but more research is needed.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Too much AKR1C3 enzyme causes resistance to finasteride by increasing testosterone.

Some women with PCOS have CYP21 mutations and IRS1 variants, but these genetic factors are not major contributors to PCOS.

Androgens may block hair growth signals, targeting this could treat hair loss.

Valproic acid increases hair count in men with hair loss.

Androgen is important in controlling stem cell differentiation, reducing fat development, and increasing lean mass.

Shorter CAG repeats in a specific gene may increase male hormone activity and symptoms like acne and excess hair in women with PCOS.

Androgens like testosterone are important hormones for both men and women, made differently in each sex and affecting the body by regulating genes and quick interactions with cell components.

Androgen Deprivation Therapy for prostate cancer often reduces sexual function but intermittent therapy may be more tolerable.

Women with androgen excess, especially those with PCOS, have a much higher risk of heart disease and stroke.

Finasteride irreversibly affects human steroid 5α-reductase 2, providing insight into its catalytic mechanism and disease-related mutations.

Testing for CAG repeat polymorphism in the androgen receptor gene is not currently recommended for managing hypogonadism.

Certain microRNAs are more common in balding areas and might be involved in male pattern baldness.

Blocking DHT production more strongly may help control advanced prostate cancer and improve quality of life.