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Androgen suppression therapy (AST) doesn't significantly lower bladder cancer risk, but using finasteride, a type of AST, might decrease the risk. AST also lessens the chance of cancer coming back but doesn't really affect survival rates. More research is needed to understand AST's benefits for different bladder cancers.

Finasteride-treated male rats' offspring had altered glucose metabolism, potentially increasing diabetes risk.

Cernitin™, a pollen extract, was found to significantly reduce pain and inflammation in rats with chronic prostatitis, suggesting it could be a useful alternative to certain anti-inflammatory drugs.

Female mice with disrupted 5α-reductase 1 had significant metabolic issues, including stress response problems, insulin resistance, liver fat buildup, and obesity.

The review suggests thorough evaluation and genetic testing for proper diagnosis and treatment of Chrousos syndrome.

Longer CAG repeats in gene linked to more severe hair loss in females.

Polycystic Ovary Syndrome is linked to higher risk of hypertension and heart disease.

Testosterone changes important cell communication proteins in pregnant rats' uteruses, which might affect pregnancy success.

Children receive less than half of recommended health care, and routine hormonal testing for female-pattern hair loss is unnecessary without other androgen excess symptoms.

Group a wide range of chemicals, not just phthalates, for assessing risks to male reproductive health.

Exercise and finasteride together improve ovary health in PCOS rats.

HSD3B1 gene variant and being overweight linked to hair loss in women with polycystic ovary syndrome.

Anabolic steroids boost muscle growth, SARMs increase muscle mass and bone density without side effects, and myostatin inhibitors block a protein that stops muscle growth, but each has potential risks.

The conclusion is that androgen receptor gene polymorphism might be a marker for polycystic ovary syndrome, but more research is needed.

Androgen receptors in the mouse brain may explain cognitive and mood changes in prostate cancer treatment.

Thyroid hormones and androgens affect gene expression in frog reproductive organs differently between males and females.

The document concludes that diagnosing PCOS in teenagers is difficult and should focus on specific hormone levels and menstrual irregularities, while also considering treatment for symptoms and related health issues.

The document concludes that research on sulfatase inhibitors should continue due to their potential in treating various diseases, despite some clinical trial failures.

Middle-aged women with PCOS often continue to experience symptoms related to high androgen levels, like unwanted hair growth.

Treating non-classic congenital adrenal hyperplasia is complex because the benefits of hormone treatment must be weighed against potential health risks.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

Certain gene variations are linked to a higher risk of severe acne, suggesting a genetic influence on the condition.

A topical treatment safely and effectively reduced acne by causing targeted cell death in sebaceous glands without side effects.

5α-reductase inhibitors help treat disorders caused by DHT and have potential for future therapies.

Hormone therapy for prostate cancer can increase heart risks, especially in men with heart conditions.

Two gene areas linked to male pattern baldness found, more research needed.

Treatments for common hair loss include minoxidil, finasteride, and hair transplantation.

The document concludes that a systematic approach is crucial to identify causes of androgen excess in women beyond the most common cause, Polycystic ovary syndrome (PCOS).

Both oral contraceptives reduced acne effectively and had similar positive effects on skin oiliness and hair growth.

The study concluded that both estrogen and androgen receptors, which decrease with age, are linked to skin aging and may be hormonally regulated.