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Research 31–60 of 1000+
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- Vitamin D deficiency in alopecia areata
- Cloning of a Functional Vitamin D Receptor from the Lamprey (Petromyzon marinus), an Ancient Vertebrate Lacking a Calcified Skeleton and Teeth
- Overexpression of Hedgehog Signaling Is Associated with Epidermal Tumor Formation in Vitamin D Receptor–Null Mice
- Does D matter? The role of vitamin D in hair disorders and hair follicle cycling
- Vitamin D and aging
- Vitamin D and calcium regulation of epidermal wound healing
- An Unusual Form of Vitamin D-Dependent Rickets in a Child: Alopecia and Marked End-Organ Hyposensitivity to Biologically Active Vitamin D
- VITAMIN D RESISTANT RICKETS WITH ALOPECIA: A FORM OF END ORGAN RESISTANCE TO 1,25 DIHYDROXY VITAMIN D
- Vitamin D-dependent rickets type II. Defective induction of 25-hydroxyvitamin D3-24-hydroxylase by 1,25-dihydroxyvitamin D3 in cultured skin fibroblasts.
- Development and progression of alopecia in the vitamin D receptor null mouse
- Hairless Suppresses Vitamin D Receptor Transactivation in Human Keratinocytes
- Vitamin D and the skin
- Novel mechanisms for the vitamin D receptor (VDR) in the skin and in skin cancer
- Topographical and developmental studies on target sites of 1,25 (OH2) vitamin D3 in skin
- Hereditary 1,25-Dihydroxyvitamin D Resistant Rickets due to a Mutation Causing Multiple Defects in Vitamin D Receptor Function
- Serum Ferritin and Vitamin D in Female Hair Loss: Do They Play a Role?
- Quantification of the Vitamin D Receptor−Coregulator Interaction
- Vitamin D and androgen receptor-targeted therapy for triple-negative breast cancer
- Targeted Expression of Human Vitamin D Receptor in the Skin Promotes the Initiation of the Postnatal Hair Follicle Cycle and Rescues the Alopecia in Vitamin D Receptor Null Mice
- Vitamin D‐dependent rickets type I and type II
- Ligand-Dependent Actions of the Vitamin D Receptor Are Required for Activation of TGF-β Signaling during the Inflammatory Response to Cutaneous Injury
- The Vitamin D Receptor Is Required for Activation of cWnt and Hedgehog Signaling in Keratinocytes
- The Vitamin D Receptor as Tumor Suppressor in Skin
- Vitamin D Concentrations are Decreased in Patients with Alopecia Areata
- Role of the vitamin D receptor in hair follicle biology
- Vitamin D Receptor, UVR, and Skin Cancer: A Potential Protective Mechanism
- The hair cycle and Vitamin D receptor
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Vitamin D-dependent rickets type II: extreme end organ resistance to 1,25-dihydroxy vitamin D3 in a patient without alopecia