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Vitamin D receptor is crucial for starting hair growth after birth.

Vitamin D and its receptor may help prevent skin cancer.

Vitamin D receptor may help protect against UV-induced skin cancer.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

People with alopecia areata often have lower vitamin D levels and are more likely to be deficient in it.

Vitamin D is important for skin health and can help treat psoriasis, atopic dermatitis, and vitiligo.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Topical vitamin D3 does not prevent hair loss from chemotherapy.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

Vitamin C helps adipose-derived stem cells grow and may support hair growth.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

Vitamin D3 and its analogues affect skin cell growth and development depending on cell density, calcium levels, and serum presence.

Vitamin D might help regulate insulin in the body, but taking Vitamin D supplements doesn't clearly prevent or improve type 2 diabetes. More research is needed.

People with alopecia areata often have lower vitamin D levels and more inflammation, suggesting vitamin D might be involved in the condition.

LEF1 interacts with Vitamin D Receptor, affecting hair follicle regeneration and this could be linked to hair loss conditions.

The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

The vitamin D receptor can work without its usual activating molecule.

Low vitamin D might be linked to certain types of hair loss, and supplements could help, but more research is needed.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

Vitamin D-dependent rickets Type II causes bone problems and hair loss, and doesn't improve with Vitamin D treatment.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Low vitamin D can worsen pediatric alopecia areata.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Topical vitamin D is useful for some skin conditions but not effective for others, and more research is needed.

Vitamin D Receptor is needed for hair growth in mice but not for skin stem cell maintenance.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D levels do not affect the risk of developing alopecia areata.