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Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

A child with a rare vitamin D-resistant condition improved with treatment.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

Combining skin tissue pathology with genetics has greatly improved the diagnosis and understanding of certain skin diseases.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

The document concludes that PCOS has a strong genetic component, but more research is needed to fully understand the specific genes involved.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Some lesser-known causes of PCOS include autoimmune issues, genetic mutations, and changes in the body's microbiome.

Nuclear hormone receptors play a significant role in skin wound healing and could lead to better treatment methods.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

People with alopecia areata often have lower vitamin D levels, which are linked to more severe and longer-lasting hair loss, but vitamin D receptor levels in the skin don't show the same pattern and don't predict treatment success.

Vitamin D Receptor is crucial for normal skin and hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

A vitamin D receptor mutation causes rickets and affects immune responses.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D made by the skin plays a role in immune defense and skin health, and more research is needed to understand its full effects.

Vitamin D is crucial for skin health and managing skin diseases.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

A boy with alopecia regrew hair using a vitamin D cream after other treatments failed.

Hair loss involves immune responses, inflammation, and disrupted signaling pathways.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.