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Research 61–90 of 1000+
- The Molecular Biology of the Vitamin D Receptor
- The Tumor Suppressor Actions of the Vitamin D Receptor in Skin
- Functional Analysis of VDR Gene Mutation R343H in A Child with Vitamin D-Resistant Rickets with Alopecia
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- Hereditary vitamin D-resistant rickets (HVDRR) owing to a heterozygous mutation in the vitamin D receptor
- Effect of a transcriptional inactive or absent vitamin D receptor on beta-cell function and glucose homeostasis in mice
- Mediator1: An Important Intermediary of Vitamin D Receptor–Regulated Epidermal Function and Hair Follicle Biology
- Dermatopathology and molecular genetics
- Genetic architecture of mouse skin inflammation and tumour susceptibility
- Deletion of Deoxyribonucleic Acid Binding Domain of the Vitamin D Receptor Abrogates Genomic and Nongenomic Functions of Vitamin D
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- A novel missense mutation in the mouse hairless gene causes irreversible hair loss: Genetic and molecular analyses of Hrm1Enu
- Analyse de la régulation du gène Hairless et identification des voies de signalisation affectées chez les mutants «bald Mill Hill».
- A Humanized Mouse Model of Hereditary 1,25-Dihydroxyvitamin D–Resistant Rickets Without Alopecia
- Hairless is a nuclear receptor corepressor essential for skin function
- 975 Antifibrogenic activities of novel vitamin D3 analogs are dependent on VDR expression in human fibroblasts
- In vivo function of VDR in gene expression-VDR knock-out mice
- Molecular Genetic and Endocrine Mechanisms of Hair Growth
- Hypomorphic mutation in the hairless gene accelerates pruritic atopic skin caused by feeding a special diet to mice
- Hairless: A nuclear receptor corepressor essential for skin function
- Genetic basis of polycystic ovary syndrome
- CRISPR/Cas9-mediated<i>VDR</i>knockout plays an essential role in the growth of dermal papilla cells through enhanced relative genes
- Experience of intravenous calcium treatment and long-term responses to treatment in a patient with hereditary vitamin D-resistant rickets resulting from a novel mutation
- Functional analyses of a novel missense and other mutations of the vitamin D receptor in association with alopecia
- The 1,25-dihydroxyvitamin D3-independent actions of the vitamin D receptor in skin
- The Vitamin D Receptor Is a Wnt Effector that Controls Hair Follicle Differentiation and Specifies Tumor Type in Adult Epidermis
- The Vitamin D Receptor Is Required for Activation of cWnt and Hedgehog Signaling in Keratinocytes
- Functional analysis of vitamin D receptor (VDR) using adenovirus vector
- Ligand-Independent Vitamin D Receptor Actions Essential for Keratinocyte Homeostasis in the Skin
- Clinical and Genetic Characterization of Tunisian Children with Hereditary 1,25-Dihydroxyvitamin D-Resistant Rickets