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The secretome from mesenchymal stem cells is a promising treatment that may repair tissue and avoid side effects of stem cell transplantation.

TRP channels in the skin are important for sensation and health, and targeting them could help treat skin disorders.

Skin has specialized touch receptors that can tell different sensations apart.

Electrospun nanofibers show promise for enhancing blood vessel growth in tissue engineering but need further research to improve their effectiveness.

Manatee whiskers are specially adapted for touch in water.

Chemotherapy can cause brain inflammation and damage, and understanding this process could help manage side effects.

Certain proteins and their receptors are more active during the growth phase of human hair and could be targeted to treat hair disorders.

Using skin stem cells and certain molecules might lead to scar-free skin healing.

Blocking interferon-gamma might help treat various autoimmune diseases.

γδ T cells help with hair growth during wound healing in mice.

Scientists found a gene in mice that causes early hearing loss.

Caffeine can damage hearing cells and affect hearing recovery after ear trauma.

Zebrafish larvae are used to study and find treatments for ear cell damage because they are easier to observe and test than mammals.

TRPV3 could be a target for treating pain, skin disorders, and hair problems, but more research is needed to create effective drugs.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

Blocking adenosine A2B receptor may prevent or treat hearing loss.

Melatonin may protect ear cells from damage caused by nicotine.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

Low Level Laser Therapy may improve noise-induced hearing loss.

Ivacaftor can protect against noise-induced hearing loss by reducing oxidative stress.

Activin A speeds up ear hair cell differentiation, while Follistatin slows it down.

Activin A and follistatin control when ear hair cells form in mice.

Activin A promotes ear hair cell development, while follistatin delays it.

Activin A and follistatin control when hair cells develop in mouse ears.

Sesn2 protects inner ear hair cells from damage by regulating certain cell survival pathways.

Rapamycin reduces age-related hearing cell loss in mice, but acarbose does not.

The conclusion is that genetic differences affect how the cochlea heals after hair cell loss, which may challenge the creation of hearing loss treatments.

Activating Nrf2 can help protect against hearing loss.