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Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

The conclusion is that different types of hair loss in dogs and cats can be cosmetic or serious, and affected animals should not be bred.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

Personalized treatment based on detailed tumor analysis successfully managed and reduced the patient's aggressive hair follicle cancer.

Some treatments can improve skin's defense against damage, but overuse may cause other skin problems.

Nrf2 helps protect skin from damage but too much can cause skin problems.

The study suggests that higher levels of SIRT1 and SIRT2 may improve overall cell health and aging processes.

Proteins like aPKC and PDGF-AA, substances like adenosine and ATP, and adipose-derived stem cells all play important roles in hair growth and health, and could potentially be used to treat hair loss and skin conditions.

Adult hair follicle cells can create new hair follicles from corneal cells with the right support.

Changing protein kinase levels in pituitary cells affects calcium flow and beta-endorphin release.

Copper treatments increase copper in all tissues, but brindled female mice accumulate much more copper in their kidneys without clinical effects, unlike brindled male mice where brain copper deficiency is clinically significant.

Prostate cancer cells can make their own androgens to activate the androgen receptor, and treatments like abiraterone may increase this ability, suggesting new therapies should target the entire steroid-making pathway.

Jumonji genes are important for development and their mutations can cause abnormalities, especially in the heart and brain.

AXR3 and SHY2 genes control the growth and timing of root hair development in plants.

The HR protein helps hair grow by blocking a hair growth inhibitor, aiding in hair follicle regeneration.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

The document concludes that assessing hair follicle damage due to cyclophosphamide in mice involves analyzing structural changes and suggests a scoring system for standardized evaluation.

Laminin-511 is crucial for early hair growth and maintaining important hair development signals.

Cystatin M/E strongly inhibits cathepsin V and cathepsin L, important for skin formation.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

Fatty acid transport protein 4 is essential for skin and hair development.

TNF family proteins are crucial for the development of skin features like hair, teeth, and mammary glands.

Drosha and Dicer are essential for hair follicle health and preventing DNA damage in skin cells.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.

Cancer development is like natural selection, involving mutated cells and environmental factors.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

AGD1 is important for root hair development in Arabidopsis, working with phosphoinositide signaling and the actin cytoskeleton.

WNT10A helps esophageal cancer cells spread and keep renewing themselves.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.