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Giving dihydrotachysterol to mother rats caused skin hardening and bone issues in their babies through milk.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

Estrogen deficiency can reduce the enzyme activity needed to activate vitamin D.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

People with advanced chronic kidney disease often have skin problems, which can be treated with various medications and procedures to improve their quality of life.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Dehydroepiandrosterone (DHEA) is a prohormone important for producing sex steroids and has potential health benefits.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Early diagnosis and individualized treatment improve outcomes for Congenital Adrenal Hyperplasia.

The document concludes that accurate diagnosis of ichthyosis is crucial for treatment and genetic advice, and ongoing research is needed for better therapies.

Vitamin D is important for skin health and may affect conditions like psoriasis and hair loss, but more research is needed to understand its role fully.

Understanding how cells die in the skin is important for treating skin diseases and preventing hair loss.

FGF signaling is a promising target for developing treatments for wounds, metabolic diseases, and cancer.

Finasteride improves hair loss in women with hyperandrogenism.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Skin problems are very common in people with end-stage kidney disease.

Mood stabilizers like lithium and anticonvulsants have side effects that can lead to patients stopping their medication, and managing these effects is important for treatment adherence.

Low iron and vitamin D levels are linked to hair loss in women.

Most patients with chronic kidney disease have skin problems, which get worse as the disease progresses, and dialysis doesn't greatly reduce these issues.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

Hormones and their receptors, especially androgens, play a key role in hair growth and disorders like baldness.

The document suggests creating a validated score to diagnose Cushing's Syndrome and considers plasma steroid profiling as a simpler diagnostic method.

Systemic glucocorticosteroids are a primary treatment for various skin conditions but require careful management due to potential side effects and relapses.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.