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Certain gene variations may increase the risk and severity of alopecia areata.

Polycystic ovary syndrome is a complex condition that affects women's mental, reproductive, and metabolic health throughout their lives.

Fat tissue stem cells may help increase hair growth.

Different harmful mutations in the CDH3 gene cause HJMD, but symptoms vary among individuals.

The research suggests that the global distribution of PCOS is likely due to historical human migration and that genes affecting PCOS may have different impacts on males and females.

Certain MC4R gene variants are linked to higher BMI in obese women with PCOS but do not cause PCOS.

New genes linked to ichthyosis were found, but there's still no cure; treatment options are improving.

Hormonal imbalances may play a role in frontal fibrosing alopecia, and antiandrogenic drugs combined with steroids are currently the most effective treatment.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Chemical exposure may contribute to the rise in atopic diseases and needs more research.

Women treated with X-ray for scalp fungus as children had a higher chance of hair loss, especially with higher radiation doses and severe fungus infections.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A man lost all his hair after stopping hepatitis C treatment and it didn't grow back.

4-O-methylhonokiol from Magnolia officinalis significantly promotes hair growth.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

The conclusion is that PCOS is caused by ovarian sensitivity to hormones and disrupted hormone control, possibly due to ovarian factors, and more research is needed.

Lowering testosterone speeds up wound healing in male mice.

Alopecia areata is likely caused by a combination of genetic factors and immune system dysfunction, and may represent different diseases with various causes.

Aromatase gene variation may increase female hair loss risk.

Liver transplantation is a viable option for children with propionic acidemia, improving quality of life and diet, but does not remove all risks and long-term brain outcomes are uncertain.

Genetic screening is crucial for accurately diagnosing APS-1 due to its varied symptoms.

New treatments for Hepatitis C show promise but need more research to confirm their safety and effectiveness for clinical use.

Certain skin conditions may indicate or increase the risk of Parkinson's disease.

New cells are added to the hair's dermal papilla during the active growth phase.

A woman's total hair loss from hepatitis C treatment grew back after stopping the medication.

A male with aromatase deficiency improved bone health with estradiol treatment.

Mutations in the WNT10A gene can cause skin, hair, teeth, and other disorders, and may also affect other areas like kidney and cancer, with potential for targeted treatments.

The document concludes that managing non-classical congenital adrenal hyperplasia in females requires personalized treatment, genetic counseling, and a team of specialists.

Different populations have distinct hair structures related to their ancestry.

ESR2 gene variations may be linked to female pattern hair loss.