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Researchers found new hair and nail genes, how hair reacts to UV, differences in white and pigmented hair growth, nerve changes in alopecia, treatments for baldness and alopecia, a toenail condition linked to a genetic disorder, and that nail fungus is more common in people with psoriasis.

A woman's hair changed to a dry, tangled texture that's hard to comb after treatment with spironolactone, suggesting the medication might cause such hair changes.

An 11-year-old child with total hair loss may have a genetic autoimmune disease, and the outlook for hair regrowth is not good.

The document concludes that various hair and scalp disorders in children have specific treatments and proper diagnosis is essential.

Alopecia areata causes varying hair loss patterns, affecting hair, nails, and possibly glands, with treatment outcomes depending on disease duration and extent.

The skin is vital for protection, temperature control, fluid balance, immunity, and sensing, with damage affecting daily life and mental health.

The conclusion is that skin and hair patterns are formed by a mix of cell activities, molecular signals, and environmental factors.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

A WNT10A gene mutation leads to ectodermal dysplasia by disrupting cell growth and differentiation.

The study concluded that PKP1 is essential for skin integrity and hair growth, and its dysfunction causes the symptoms of ectodermal dysplasia/skin fragility syndrome.

The EDAR gene mutation leads to thinner and more deformed hair shafts.

Different ectodermal organs like hair and feathers regenerate differently, with specific stem cells and signals involved in their growth and response to the environment.

The HoxC gene cluster and its enhancers are essential for developing hair and nails in mammals.

Mice without certain skin proteins had abnormal skin and hair development.

Researchers found genetic mutations causing hypohidrotic ectodermal dysplasia in 88% of studied patients and identified new mutations and genetic variations affecting the disease.

Scientists now better understand the genetics of hypohidrotic ectodermal dysplasia, leading to more accurate diagnoses and potential new treatments.

Examining nail biopsies is useful for diagnosing nail diseases.

Giving a special protein to dogs with a certain genetic disease improved their symptoms but didn't help with hair growth.

Mutations in the KRT85 gene cause hair and nail problems.

People with X-linked hypohidrotic ectodermal dysplasia have no sweat ducts and less, thinner hair.

Blocking the FGF5 gene in sheep leads to more fine wool and active hair follicles due to changes in certain cell signaling pathways.

AIRE-deficient rats developed severe autoimmune disease similar to APECED, useful for testing treatments.

A mother's diet at conception can cause lasting genetic changes in her child.

Epigenetic changes control how adult stem cells work and can lead to diseases like cancer if they go wrong.

Ectodysplasin inhibits Wnt signaling to help form hair follicles.

The study found that the protein Dkk4 helps regulate hair growth by controlling Wnt signaling in mice.

Human stem cells can help form hair follicles in mice.

Future research on ectodysplasin should explore its role in diseases, stem cells, and evolution, and continue developing treatments for genetic disorders like hypohidrotic ectodermal dysplasia.

DNA methylation changes are linked to hair growth cycles in goats.

Scaffold-based strategies show promise for regenerating hair follicles and teeth but need more research for clinical use.