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- Anticytokine therapy, particularly anti-IFN-γ, in Th1-mediated autoimmune diseases
- Inheritance-Specific Dysregulation of Th1- and Th17-Associated Cytokines in Alopecia Areata
- The Role of Serum Th1, Th2, and Th17 Cytokines in Patients with Alopecia Areata: Clinical Implications
- Class IIa HDAC4 and HDAC7 cooperatively regulate gene transcription in Th17 cell differentiation
- Regulatory T cell dysfunction and immunotherapeutic breakthroughs in type 1 diabetes
- Th1 effector CD4 T cells rely on IFN-γ production to induce alopecia areata
- T <sub>H</sub> 1 effector CD4 T cells rely on IFN-γ production to induce alopecia areata
- Anti-TNF antibody-induced psoriasiform skin lesions in patients with inflammatory bowel disease are characterised by interferon-γ-expressing Th1 cells and IL-17A/IL-22-expressing Th17 cells and respond to anti-IL-12/IL-23 antibody treatment
- Alopecia areata profiling shows TH1, TH2, and IL-23 cytokine activation without parallel TH17/TH22 skewing
- An integrated model of alopecia areata biomarkers highlights both TH1 and TH2 upregulation
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