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Dermal papillae cells, important for hair growth, come from multiple cell lines and can be formed by skin cells, regardless of their origin or hair cycle phase. These cells rarely divide, but their ability to shape tissue may contribute to their efficiency in inducing hair growth.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

The demineralized bone matrix scaffold is better for cell attachment than the mineralized bone allograft.

Genomic profiling for myeloid cancers can find important inherited mutations, but it's challenging when these mutations aren't related to the patient's symptoms.

Human skin and hair color variation is mainly due to melanin produced by a few genes, with melanin protecting against sun damage.

Birt–Hogg–Dubé syndrome is a rare genetic condition causing skin lesions, lung cysts, and a higher chance of kidney cancer.

Cutaneous Lymphadenoma is a unique skin tumor with specific protein markers and common gene mutations that may cause continuous cell growth.

The research linked PLCD1 gene variants to the development of trichilemmal cysts.

New techniques have enhanced our understanding of how stem cells function and the role of mutations in aging tissues, which may influence future cancer therapies.

Beige and leaden pigment genes act within melanocytes, affecting pigment patterns.

A mouse gene mutation increases the risk of skin cancer.

Key genes can rewire networks, changing skin appendage types.

The research found that the gene activity in mouse skin stem cells changes significantly as they age.

The conclusion is that the nuclear lamina and LINC complex in skin cells respond to mechanical signals, affecting gene expression and cell differentiation, which is important for skin health and can impact skin diseases.

Increasing mir-302 turns human hair cells into stem cells by changing gene regulation and demethylation.

Deleting the EDAR gene in Cashmere goats affects genes and proteins related to hair growth.

Knocking out certain genes in mice helps understand skin and hair growth problems.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Stress increases certain chemicals in the skin and nerves, which might worsen skin conditions.

A new method can detect mutations in mice by observing changes in hair follicle cells.

Skin-derived precursors in hair follicles come from different origins but function similarly.

Scientists created stem cells that can grow hair and skin.

Basal cell carcinoma mostly starts from cells in the upper skin layers, not hair follicle stem cells.

Epigenetic mechanisms control skin development by regulating gene expression.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

A new genetic change in the keratin 10 gene caused a skin condition called ichthyosis hystrix in a father and his daughter.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Meis2 is essential for touch sensation and proper nerve connection to touch receptors in certain skin areas of mice.