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People who carry the 21-Hydroxylase Deficiency gene are not more likely to have excessive male hormone levels.

Women with nonclassic congenital adrenal hyperplasia experience more sexual dysfunction and distress.

Certain genetic variants impair enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

Certain genetic variants reduce enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

CAH is a genetic disorder affecting cortisol production and causing hormonal imbalances, with treatment and diagnosis varying by form and symptoms.

Most women with polycystic ovary syndrome (PCOS) have menstrual issues, infertility, acne, and excessive body hair, with the most common type being the non-androgenic phenotype.

Children with classic congenital adrenal hyperplasia have thicker heart fat and are at higher risk for heart problems and early atherosclerosis.

11α-Hydroxyprogesterone is changed into different substances by certain enzymes and may play a role in prostate cancer.

Most adrenal cortex tumors are benign and non-secreting, but proper diagnosis and treatment are important due to the rare possibility of cancer.

The conclusion is that while oral contraceptive pills are effective for PCOS-related high androgen levels, new treatments with fewer side effects are needed.

Insulin resistance is linked to PCOS and can lead to other health issues, but treatments like metformin can help manage symptoms.

Blocking CRF1 receptors improved male hormone levels and reduced testicular tumor size in men with a specific adrenal condition.

Hormones, especially testosterone and DHT, are key for penis development and function, and testosterone therapy may help with erectile dysfunction in those with low levels.

Children with classic congenital adrenal hyperplasia have thicker heart fat and more heart and blood vessel risk factors, especially if their condition is not well-controlled.

Insulin resistance is a key factor in polycystic ovary syndrome, but genetics may also contribute.

Women with androgen excess, especially those with PCOS, have a much higher risk of heart disease and stroke.

The conclusion is that accurately identifying the cause of high androgen levels in women with PCOS is crucial and requires specific tests.

Experts recommend using evidence-based methods to diagnose and treat hirsutism, focusing on symptoms and underlying causes.

Acne is a chronic disease linked to various systemic conditions and has significant psychological and social effects.

Rare ULBP3 gene changes may raise the risk of Alopecia areata, a certain FAS gene deletion could cause a dysfunctional protein in an immune disorder, and having one copy of a specific genetic deletion is okay, but two copies cause sickle cell disease.

Insulin resistance is linked to high male hormone levels in women, often seen in those with PCOS.

Diagnosing simple-virilizing congenital adrenal hyperplasia can be difficult and requires thorough testing and expert advice.

Flutamide works better than finasteride for hirsutism, and combining both is slightly better but not much.

More research is needed to understand how testosterone is maintained in adult males.

AMH levels can't reliably tell the difference between LOCAH and all types of PCOS in women with excessive hair growth.

Hirsutism is mainly caused by high androgen levels or sensitivity, with PCOS being the most common cause.

Some women with PCOS have CYP21 mutations and IRS1 variants, but these genetic factors are not major contributors to PCOS.

The document concludes that treatments for female hair loss and excess body hair are available, but managing expectations is important.

The article suggests renaming Polycystic Ovarian Syndrome (PCOS) to "Hyperandrogenic Persistent Ovulatory Dysfunction Syndrome" (HA-PODS) for accuracy and consistency, but no final decision was made.

Eating a Mediterranean diet and taking certain supplements may improve symptoms of PCOS.