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Research 30 of 1000+
- Bridging Personal and Population in Excitability Diseases: Will Studies of Rare Diseases Bring Generalizable Mechanisms From Monogenic Channelopathies?
- Dermatological diseases from a genetic perspective
- What does acne genetics teach us about disease pathogenesis?
- Unveiling the Roots of Monogenic Genodermatoses: Genotrichoses as a Paradigm
- Computational derivation of a molecular framework for hair follicle biology from disease genes
- Inherited Disorders of Corneocyte Proteins
- Molecular Genetics of Alopecias
- Identification and characterization of genes for skin and hair disorders
- Type II Keratins Are Phosphorylated on a Unique Motif during Stress and Mitosis in Tissues and Cultured Cells
- The Genetics of Human Skin Disease
- JAK1 gain-of-function variant causes alopecia areata, atopic dermatitis, and autoimmune thyroid disease
- Corneodesmosomes and corneodesmosin: from the stratum corneum cohesion to the pathophysiology of genodermatoses
- E-Poster
- TRANSTORNOS DISSOCIATIVOS EM PACIENTES VÍTIMAS DE ABUSO SEXUAL NA INFÂNCIA
- Skin diseases in consequence of endocrine alterations
- Genome-wide association study of skin complex diseases
- Evolving approaches to profiling the microbiome in skin disease
- Fibroblast heterogeneity: implications for human disease
- THE DISEASE SPECTRUM, EPIDEMIOLOGY, AND ETIOLOGY OF TOXIC-SHOCK SYNDROME
- Male fertility and skin diseases
- Insulin Resistance and Skin Diseases
- Recent Advances in Drug Design and Drug Discovery for Androgen- Dependent Diseases
- The role of protein lactylation in skin diseases: from molecular mechanisms to potential therapeutics
- Polycystic Ovarian Disease: Current Insights and therapeutic Strategies
- Structures of three polycystic kidney disease-like domains from<i>Clostridium histolyticum</i>collagenases ColG and ColH
- PAK4 signaling in health and disease: defining the PAK4–CREB axis
- Androgens and Parkinson’s disease: the role in humans and in experiment
- Cachexia and graft-vs.-host-disease-type skin changes in keratin promoter-driven TNF alpha transgenic mice.
- K <sub>ATP</sub> Channels and Cardiovascular Disease
- Kin‐cohort analysis of <i>LRRK2</i>‐G2019S penetrance in Parkinson's disease