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Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

The vitamin D receptor is crucial for bone health and affects various body systems, with mutations potentially leading to disease.

FGF signaling is a promising target for developing treatments for wounds, metabolic diseases, and cancer.

The document reports the first Brazilian case of a girl with Becker nevus syndrome, characterized by skin changes, breast underdevelopment, and scoliosis.

Whale genes show changes that help them live in water, like less hair and better flippers.

Vitamin D and calcium are essential for normal hair growth.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

Two sisters with rickets and hair loss had a genetic issue with vitamin D processing, and only improved when given phosphorus supplements.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Vitamin D and its receptor are essential for hair growth, cell regulation, immune function, and heart health.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

A child with a rare vitamin D-resistant condition improved with treatment.

Early diagnosis and treatment of hereditary vitamin D-resistant rickets (HVDRR) are crucial to prevent growth issues and other health problems.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

A vitamin D receptor mutation causes rickets and affects immune responses.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Vitamin D is important for bones, hair, blood pressure, and breast development.

Six genetic conditions are often linked to complete scalp hair loss in children.

Mice without the vitamin D receptor have bone issues and other health problems, suggesting vitamin D is important for preventing various diseases in humans.

Low vitamin D might be linked to certain types of hair loss, and supplements could help, but more research is needed.

Vitamin D3 may protect rat hair follicles from radiation damage.