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The conclusion is that individual differences in COVID-19 severity are influenced by factors like age, sex, race, and genetics, which are important for personalized medicine.

Some patients with mycosis fungoides or Sézary syndrome experience hair loss, which may be similar to alopecia areata or linked to skin lesions, possibly due to abnormal T cells, and bexarotene can help treat it.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Genetic factors alone might not cause pemphigus vulgaris; other factors like birth complications and puberty may trigger it.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

The document concludes that pregnancy and cancer share immune evasion tactics, but more research is needed before using checkpoint blockade immunotherapy in pregnant cancer patients to avoid harm to the placenta.

A woman's hair loss worsened after starting hepatitis C treatment due to immune changes in her hair follicles.

Small amounts of DNA can be found in the hair shaft, especially near the root, but it decreases with hair treatments and washing.

Some cancer treatments can cause abnormal fine hair growth.

The cause of Frontal fibrosing alopecia, a type of hair loss, is complex, likely involving immune responses and genetics, but is not fully understood.

Frontal Fibrosing Alopecia is a hair loss condition mainly affecting postmenopausal women, with unclear causes and various clinical patterns.

Down Syndrome patients with Alopecia Areata often have hypothyroidism and specific immune abnormalities.

Alopecia areata is an autoimmune disease with genetic links, treatable with certain medications, and can affect mental health.

Women with excessive hair growth or polycystic ovary disease may more often carry a gene variant for 21 hydroxylase deficiency.

The exact cause of frontal fibrosing alopecia is unknown, but it's not likely due to sunscreen.

Inflammatory acne damages skin stem cells and reduces their growth, leading to atrophic acne scars.

Chronic bacterial infections of hair follicles can cause ongoing skin inflammation.

A woman with lupus also developed a severe skin condition linked to a genetic factor.

The patient with Ankylosing Spondylitis had shoulder joint damage and bone changes.

Different genes are linked to various types of hair loss.

Alopecia areata, a type of hair loss, may be passed through T cells and has genetic links, while treatments vary in effectiveness. Male pattern baldness can be treated with finasteride and is influenced by androgens in hair follicles.

Tebentafusp-tebn improves survival rates in uveal melanoma patients but has common side effects like rash and fatigue.

Herpes gestationis is linked to certain antigens, atopic eczema affects T cell populations and may be eased by breastfeeding, higher doses of anti-androgen treatment can improve androgenic alopecia, topical minoxidil increases hair thickness, long-term methotrexate therapy can cause liver fibrosis in psoriasis patients, and Lichen Sclerosus et Atrophicus patients aren't at higher risk for autoimmune disorders.

A mother and son both had ulcerative colitis and alopecia, suggesting a genetic link in autoimmune disorders and successful treatment with cyclosporine.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

A man had two rare autoimmune diseases that might be connected.

Oral contraceptive use may increase the risk of frontal fibrosing alopecia in women with a specific CYP1B1 gene variant.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.