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Higher miR-34a levels and the A variant of the MIR-34A gene are linked to increased risk and severity of alopecia areata.

Genomic profiling for myeloid cancers can find important inherited mutations, but it's challenging when these mutations aren't related to the patient's symptoms.

The Wnt/β-catenin pathway is important for body functions and diseases, and targeting it may treat conditions like cancer, but with safety challenges.

Vitamin D is important for skin health and can help treat psoriasis, atopic dermatitis, and vitiligo.

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Blocking the ATX-LPA pathway may improve insulin sensitivity and mitochondrial function in obesity.

JAK inhibitors show promise in effectively treating hair loss from alopecia areata.

Blocking certain immune signals can reduce skin damage from radiation therapy.

Portraits show women's hairlines have moved forward over time, suggesting changes in the occurrence of frontal hair loss.

Scientists are still unsure what triggers the immune system to attack hair follicles in Alopecia areata.

The 2015 Hair Research Congress concluded that stem cells, maraviroc, and simvastatin could potentially treat Alopecia Areata, topical minoxidil, finasteride, and steroids could treat Frontal Fibrosing Alopecia, and PTGDR2 antagonists could also treat alopecia. They also found that low-level light therapy could help with hair loss, a robotic device could assist in hair extraction, and nutrition could aid hair growth. They suggested that Alopecia Areata is an inflammatory disorder, not a single disease, indicating a need for personalized treatments.

A mother and daughter with similar hair loss conditions and identical HLA types suggest a genetic link between the conditions.

Most women with ankylosing spondylitis tested positive for HLA-B27, suggesting it's useful for diagnosis.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.

Severe alopecia areata involves higher levels of certain immune cells, which can be normalized with betamethasone.

Alopecia areata, a common autoimmune hair loss condition, often runs in families.

Alopecia areata is an autoimmune disease where T cells attack hair follicles.

A gluten-free diet significantly improves skin and health issues in people with dermatitis herpetiformis, a skin condition linked to celiac disease.

FOXN1 gene mutations cause a rare, severe immune disease treatable with cell or tissue transplants.

Thymic transplantation normalized some T-cells but not others, maintaining immune function.

Patients with Alopecia areata have fewer specific immune cells that normally regulate the immune system, which may contribute to the condition.

Genetic factors alone might not cause pemphigus vulgaris; other factors like birth complications and puberty may trigger it.

The cause of alopecia areata is likely a mix of genetics, immune system issues, and environmental factors, with more research needed to understand it fully.

A woman's hair loss worsened after starting hepatitis C treatment due to immune changes in her hair follicles.

Small amounts of DNA can be found in the hair shaft, especially near the root, but it decreases with hair treatments and washing.

Some cancer treatments can cause abnormal fine hair growth.

Down Syndrome patients with Alopecia Areata often have hypothyroidism and specific immune abnormalities.

The exact cause of frontal fibrosing alopecia is unknown, but it's not likely due to sunscreen.

Inflammatory acne damages skin stem cells and reduces their growth, leading to atrophic acne scars.