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Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

The more lipophilic the progesterone derivative, the better it binds to androgen receptors and has antiandrogenic effects.

Antiandrogen therapy helps treat genetic hair loss.

A gene called HDAC9 might be a new factor in male-pattern baldness.

Mice with human gene experienced hair loss when treated with DHT.

A patient with a larger than usual genetic mutation had a broader range of symptoms for a muscle disease.

Testosterone affects hair follicles differently across body sites, with beard hair follicles showing more activity of a specific enzyme and presence of androgen receptors compared to scalp hair.

Certain inhibitors can potentially treat prostate cancer and other hormone-dependent conditions by controlling sex hormone levels in cells.

Hormones and genetics play key roles in male and female baldness, which can affect mental health and may be linked to other health issues.

The document concludes that blocking the internal pathways that create androgens might help treat cancers that depend on sex hormones.

Minoxidil can reduce functions related to androgen receptors.

Hair loss from genetics and hormones can be treated with drugs or surgery.

Men have worse COVID-19 outcomes than women due to genetic and hormonal differences.

Cyproterone acetate plus ethinyl estradiol is generally more effective in treating hirsutism, but consider side effects and patient characteristics.

Nonclassic congenital adrenal hyperplasia is a genetic disorder causing hormone imbalances, affecting fertility and requiring personalized treatment.

CAH is a genetic disorder affecting cortisol production and causing hormonal imbalances, with treatment and diagnosis varying by form and symptoms.

Androgens increase norepinephrine release, promoting smooth muscle growth in male sex organs, which may contribute to benign prostatic hypertrophy.

Jagged1 and Epidermal Growth Factor together significantly increased hair growth in mice with androgen-suppressed hair.

Clascoterone may be an effective topical treatment for hair loss.

Excessive hair growth in women, often from high androgen levels, is usually caused by PCOS, and can be treated with hair removal, medication, and possibly weight loss.

Male pattern baldness may be caused by scalp pressure on hair follicles, which increases with age and leads to a cycle of hair loss. This process is not directly determined by genes.

Testosterone can slow down hair growth when combined with certain cells from bald scalps, and this effect can be blocked by an androgen receptor blocker.

Androgens, male hormones, play a role in both men and women's health, and testosterone therapy can help women with low levels, but it's not suitable for pregnant or lactating women, or those with certain conditions.

Early hormones shape sex-specific differences in rat glands.

Understanding how androgens and their receptors work can lead to improved treatments for skin diseases.

Hirsutism is mainly caused by high androgen levels or sensitivity, with PCOS being the most common cause.

The oral contraceptive ethinylestradiol/chlormadinone acetate is effective in reducing acne and improving other skin conditions related to high androgen levels.

Excessive hair growth in women often has no known cause and is not linked to race or other hormonal symptoms.

Certain medications and maintaining adequate iron levels can help manage women's hair loss.

Male-pattern hair loss is largely influenced by genetics, with key genes identified.