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    Glossary c-Jun N-terminal kinase

    regulates cellular stress responses and apoptosis

    c-Jun N-terminal kinase (JNK) is a type of protein kinase that plays a crucial role in regulating various cellular processes, including inflammation, cell growth, and apoptosis (programmed cell death). It is activated in response to stress signals, such as UV radiation or cytokines, and can influence gene expression by modifying the activity of specific transcription factors like c-Jun.

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    1. Protective effect of DA-9401 in finasteride-induced apoptosis in rat testis: inositol requiring kinase 1 and c-Jun N-terminal kinase pathway Drug Design Development and Therapy · 2017 · 18 citations
    2. Protective effect of DA-9401 in finasteride-induced apoptosis in rat testis: inositol requiring kinase 1 and c-Jun N-terminal kinase pathway DOAJ (DOAJ: Directory of Open Access Journals) · 2017
    3. 446 Protective effect of DA-9401 in finasteride-induced apoptosis in rat testis: inositol requiring kinase 1 and c-Jun N-terminal kinase pathway The Journal of Sexual Medicine · 2018
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    6. LB1076 Using the frog embryonic epidermis as a model to study desmosome function during development Journal of Investigative Dermatology · 2019
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    8. The Molecular Mechanism Underlying the Proliferating and Preconditioning Effect of Vitamin C on Adipose-Derived Stem Cells Stem Cells and Development · 2014 · 42 citations
    9. Enzymes British Journal of Pharmacology · 2008
    10. Context-dependent effect of sPLA2-IIA induced proliferation on murine hair follicle stem cells and human epithelial cancer EBioMedicine · 2019 · 13 citations
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