The development of universal alopecia during therapy with TNF-α inhibitors in patients with ankylosing spondylitis: description of three cases

The study concluded that immune cells attacking hair follicles cause hair loss in alopecia, with genetics and environment also playing a role, and highlighted the potential of certain treatments.

Alopecia areata is likely an autoimmune disease with unclear triggers, involving various immune cells and molecules, and currently has no cure.

A woman with rheumatoid arthritis lost all her hair during adalimumab treatment, and stopping the drug didn't help.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Tofacitinib is effective and safe for severe hair loss, but full regrowth is less likely after 10 years of hair loss.

A woman with severe hair loss was successfully treated with the drug adalimumab.

Alopecia areata can be reversed by JAK inhibitors, promoting hair regrowth.

Alopecia Areata might be linked to Crohn's disease.

Anti-TNF-α therapy may increase the risk of developing alopecia areata, especially in those with a history of autoimmune disease.

Half of people with Alopecia Areata may see hair regrowth within a year without treatment, but recovery is unpredictable.

Alopecia areata often starts before age 20, is more common in women, and may have a genetic link with other autoimmune diseases.