Search
for

    Glossary CXCR3

    receptor on immune cells guiding them to inflammation sites

    CXCR3 is a receptor found on the surface of certain immune cells, such as T cells. It plays a crucial role in directing these cells to sites of inflammation or injury by binding to specific signaling molecules called chemokines. This receptor is important in various immune responses and has been implicated in conditions like autoimmune diseases and alopecia, where the immune system mistakenly attacks the body's own tissues.

    Related Terms

    Research 10 of 731

    1. PD-1+CXCR5−CD4+T cells are correlated with the severity of systemic lupus erythematosus Rheumatology · 2019 · 71 citations
    2. CXCR3 Blockade Inhibits T Cell Migration into the Skin and Prevents Development of Alopecia Areata Journal of Immunology · 2016 · 64 citations
    3. Author response: Neutrophils promote CXCR3-dependent itch in the development of atopic dermatitis 2019 · 1 citations
    4. Decision letter: Neutrophils promote CXCR3-dependent itch in the development of atopic dermatitis 2019
    5. Regulatory T cells in skin utilize the Cxcr4-Cxcl12 axis to promote hair follicle regeneration Cell Reports · 2025
    6. CXCL12 inhibits hair growth through CXCR4 Biomedicine & Pharmacotherapy · 2022
    7. Inducible Cutaneous Inflammation Reveals a Protumorigenic Role for Keratinocyte CXCR2 in Skin Carcinogenesis Cancer Research · 2008 · 70 citations
    8. Preconditioning with far-infrared irradiation enhances proliferation, cell survival, and migration of rat bone marrow-derived stem cells via CXCR4-ERK pathways Scientific Reports · 2017 · 44 citations
    9. Plerixafor Engages β-Arrestin-Dependent CXCR4 Signaling to Promote Melanogenesis via β-Catenin-MITF Activation Current Issues in Molecular Biology · 2026
    10. Plerixafor Biases CXCR4 Signaling Through β-Arrestin to Promote Melanogenesis via β-Catenin–MITF Activation Preprints.org · 2026
    All research results →

    Learn

    — no results

    Try a deeper search in learn →

    Community

    — no results

    Try a deeper search in community →