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Research 10 of 404
- Impaired wound healing in transgenic mice overexpressing the activin antagonist follistatin in the epidermis
- Activin Controls Skin Morphogenesis and Wound Repair Predominantly via Stromal Cells and in a Concentration-Dependent Manner via Keratinocytes
- Control of pelage hair follicle development and cycling by complex interactions between follistatin and activin
- The roles of activins in repair processes of the skin and the brain
- Conditional Activin Receptor Type 1B (Acvr1b) Knockout Mice Reveal Hair Loss Abnormality
- The activin binding proteins follistatin and follistatin-related protein are differentially regulated in vitro and during cutaneous wound repair
- The role of activins and follistatins in skin and hair follicle development and function
- Activin B Promotes BMSC-Mediated Cutaneous Wound Healing by Regulating Cell Migration via the JNK—ERK Signaling Pathway
- GLI2-specific Transcriptional Activation of the Bone Morphogenetic Protein/Activin Antagonist Follistatin in Human Epidermal Cells
- Activin B Promotes Epithelial Wound Healing In Vivo through RhoA-JNK Signaling Pathway
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