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    Glossary Wnt10b

    protein crucial for activating hair follicle stem cells

    Wnt10b is a protein that plays a crucial role in the regulation of hair growth and development. It is part of the Wnt signaling pathway, which is essential for various cellular processes, including cell proliferation, differentiation, and migration. In the context of hair, Wnt10b helps to activate hair follicle stem cells, promoting the growth of new hair.

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    Research 10 of 929

    1. WNT10A Mutations Are a Frequent Cause of a Broad Spectrum of Ectodermal Dysplasias with Sex-Biased Manifestation Pattern in Heterozygotes American journal of human genetics · 2009 · 197 citations
    2. WNT10A mutation causes ectodermal dysplasia by impairing progenitor cell proliferation and KLF4-mediated differentiation Nature Communications · 2017 · 96 citations
    3. Targeting Wnt/β-Catenin Pathway for Developing Therapies for Hair Loss International Journal of Molecular Sciences · 2020 · 86 citations
    4. Adenovirus-Mediated Wnt10b Overexpression Induces Hair Follicle Regeneration Journal of Investigative Dermatology · 2012 · 77 citations
    5. Treatment of MSCs with Wnt1a-conditioned medium activates DP cells and promotes hair follicle regrowth Scientific Reports · 2014 · 61 citations
    6. WNT10A promotes an invasive and self-renewing phenotype in esophageal squamous cell carcinoma Carcinogenesis · 2015 · 55 citations
    7. The balance of Bmp6 and Wnt10b regulates the telogen-anagen transition of hair follicles Cell Communication and Signaling · 2019 · 35 citations
    8. Wnt10b Promotes Differentiation of Mouse Hair Follicle Melanocytes International Journal of Medical Sciences · 2013 · 29 citations
    9. The role of WNT10B in physiology and disease: A 10-year update Frontiers in Cell and Developmental Biology · 2023 · 27 citations
    10. Wnt1a maintains characteristics of dermal papilla cells that induce mouse hair regeneration in a 3D preculture system Journal of Tissue Engineering and Regenerative Medicine · 2015 · 23 citations
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