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- dsRNA Released by Tissue Damage Activates TLR3 to Drive Skin Regeneration
- The L412F variant of Toll-like receptor 3 (TLR3) is associated with cutaneous candidiasis, increased susceptibility to cytomegalovirus, and autoimmunity
- Noncoding dsRNA induces retinoic acid synthesis to stimulate hair follicle regeneration via TLR3
- Hypothesis: Wound‐induced TLR3 activation stimulates endogenous retinoic acid synthesis and signalling during regeneration
- Activation of TLR3 Enhance Stemness and Immunomodulatory Properties of Periodontal Ligament Stem Cells (PDLSCs)
- TLR3 Activation Stimulates Autophagy Gene Expression in Keratinocytes
- TLR3 signaling modulates immunomodulatory property of human periodontal ligament stem cells.
- Three-dimensional gelatin sponge culture potentiates MSC secretome to enhance full-thickness wound healing and induce hair follicle neogenesis via Wnt/β-catenin and TLR3/STAT3 activation in rats
- Toll-Like Receptor Function in Acute Wounds
- Through the lens of hair follicle neogenesis, a new focus on mechanisms of skin regeneration after wounding
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