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The 2003 consensus updated PCOS diagnosis criteria and highlighted increased risks of diabetes and heart disease for those affected.

The 2003 consensus updated PCOS diagnosis criteria and linked PCOS to higher risks of diabetes and heart problems, recommending lifestyle changes to lower these risks.

The report concludes that PCOS is mainly a condition of excess male hormones and its definition may change as new information is discovered.

Most women with excess male hormones have Polycystic Ovary Syndrome, and hormonal therapy can improve symptoms but may cause side effects.

Insulin resistance is a key factor in polycystic ovary syndrome, but genetics may also contribute.

The guidelines suggest a detailed approach to diagnosing and treating lupus, with a focus on regular check-ups, personalized medicine, and a range of drug options for different cases.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

Male hormones affect COVID-19 severity and certain drugs targeting these hormones could help reduce the risk.

Fat-derived stem cells show promise for skin repair and reducing aging signs but need more research for consistent results.

The conclusion is that PCOS is caused by ovarian sensitivity to hormones and disrupted hormone control, possibly due to ovarian factors, and more research is needed.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

IL-2 treatment causes skin eruptions and other reversible side effects, and may play a role in psoriasis.

The research found that Atypical Progeroid Syndrome has unique symptoms and is not caused by the buildup of a certain mutant protein.

South Asian women with PCOS are more likely to have metabolic issues and central obesity, and simple measures like waist size and blood pressure can help identify these risks early.

Researchers identified genes linked to coat color, body size, cashmere production, and high altitude adaptation in goats.

Six new genetic regions linked to early hair loss also connect to Parkinson's disease and prostate cancer, possibly leading to new treatments.

Minoxidil boosts hair growth by targeting adenosine and possibly sulfonylurea receptor 2B.

Topical drugs and near-infrared light therapy show potential for treating alopecia.

FFA's causes may include environmental triggers and genetic factors.

β-Catenin signaling is involved in brain cell growth after injury and could be a therapy target.

Researchers found 63 genes linked to male-pattern baldness, which could help in understanding its biology and developing new treatments.

Male pattern baldness is mostly inherited, involves many genes, and is linked to other traits like early puberty and strong bones.

Genes and hormones cause hair loss, with four genes contributing equally.

A gene in Arabidopsis thaliana, AtPRPL1, affects root hair length but not cell wall composition.

Minoxidil improves blood flow and vessel flexibility, potentially helping with vascular stiffness.

Mutations in the WNT10A gene can cause skin, hair, teeth, and other disorders, and may also affect other areas like kidney and cancer, with potential for targeted treatments.

Enzyme replacement therapy improved multiple symptoms of homocystinuria in mice.

The ITGB6 gene is important for tissue repair and hair growth, and mutations can lead to enamel defects and other health issues.

Glutathione helps Arabidopsis roots adapt to low phosphate by regulating a specific growth pathway.

The document concludes that lifestyle changes and medical treatments can significantly reduce symptoms of Hidradenitis Suppurativa, a chronic skin condition.