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Research 30 of 309
- Ectodysplasin-A2 induces dickkopf 1 expression in human balding dermal papilla cells overexpressing the ectodysplasin A2 receptor
- Epidermal patterning and induction of different hair types during mouse embryonic development
- Lgr5 Stem Cells and Potential Treatments for Hair Loss
- Transcriptome Reveals Long Non-coding RNAs and mRNAs Involved in Primary Wool Follicle Induction in Carpet Sheep Fetal Skin
- Genetic and molecular aspects of androgenetic alopecia
- Identification of Ectodysplasin Target Genes Reveals the Involvement of Chemokines in Hair Development
- p53‐dependent transcriptional regulation of EDA2R and its involvement in chemotherapy‐induced hair loss
- Dermal β-catenin activity in response to epidermal Wnt ligands is required for fibroblast proliferation and hair follicle initiation
- Recent positive selection of a human androgen receptor/ectodysplasin A2 receptor haplotype and its relationship to male pattern baldness
- Identification of dkk4 as a target of Eda-A1/Edar pathway reveals an unexpected role of ectodysplasin as inhibitor of Wnt signalling in ectodermal placodes
- Coordinating the Sculptors: Ectodysplasin-A (EDA) Signaling Cross-Talk with Skeletogenic Pathways
- Transcriptome sequencing reveals the expression profiles of lncRNAs and mRNAs in goat skin tissues with different types of wool coats
- Feather arrays are patterned by interacting signalling and cell density waves
- Molecular biology of hair morphogenesis: Development and cycling
- Hormonal regulation in male androgenetic alopecia—Sex hormones and beyond: Evidence from recent genetic studies
- Label Retaining Cells (LRCs) with Myoepithelial Characteristic from the Proximal Acinar Region Define Stem Cells in the Sweat Gland
- Use of genetics in the prediction of success in male pattern hair loss therapy and mechanistic studies
- Edar Signaling in the Control of Hair Follicle Development
- miR-29a-5p Inhibits Prenatal Hair Placode Formation Through Targeting EDAR by ceRNA Regulatory Network
- Genetics of Alopecia
- A replication study confirmed the EDAR gene to be a major contributor to population differentiation regarding head hair thickness in Asia
- Genomic Markers and Personalized Medicine in Androgenetic Alopecia: A Comprehensive Review
- A new mutation resulting in the truncation of the TRAF6-interacting domain of XEDAR: a possible novel cause of hypohidrotic ectodermal dysplasia: Figure 1
- Hair Follicle Morphogenesis During Embryogenesis, Neogenesis, and Organogenesis
- Mesenchymal–epithelial interactions during hair follicle morphogenesis and cycling
- Ectodysplasin A (EDA) Signaling: From Skin Appendage to Multiple Diseases
- KGF and EGF signalling block hair follicle induction and promote interfollicular epidermal fate in developing mouse skin
- The Roles of EDA2R in Ageing and Disease
- Increased risk of chronic fatigue and hair loss following COVID-19 in individuals with hypohidrotic ectodermal dysplasia
- BMP signaling in the control of skin development and hair follicle growth