In Vitro Suppression of Human Lymphocyte Activity by Minoxidil

January 1985 in “ International Journal of Dermatology ”

Gillian M.P. Galbraith, Bruce H. Thiers

minoxidil immune-suppressive human lymphocytes pattern baldness alopecia areata Rogaine

TLDR Minoxidil may help hair growth by stopping immune system attacks on hair follicles.

This 1985 study found that minoxidil has immune-suppressive effects on human lymphocytes in vitro, suggesting that it may work by suppressing the immune system's attack on hair follicles in individuals with pattern baldness or alopecia areata.

View this study on doi.org →

Discuss this study in the Community →

Research cited in this study

5 / 5 results

research Alopecia Areata Treated With Topical Minoxidil

16 citations, April 1984 in “Archives of Dermatology”

Topical minoxidil can help hair regrowth in alopecia areata patients, but maintaining the growth after stopping treatment is inconsistent.

research Effect of Minoxidil on Cultured Keratinocytes

72 citations, December 1983 in “Journal of Investigative Dermatology”

Minoxidil helps hair cells live longer and grow longer.

research Topical Minoxidil in the Treatment of Alopecia Areata

80 citations, October 1983 in “BMJ”

Minoxidil helps hair regrowth in alopecia patients, with 16 having good results and no side effects.

research Minoxidil: A Review of Its Pharmacological Properties and Therapeutic Use

96 citations, October 1981 in “Drugs”

Minoxidil effectively treats severe hypertension but may cause side effects, so careful monitoring is needed.

research Hypertrichosis Due to Minoxidil

45 citations, November 1979 in “British Journal of Dermatology”

Minoxidil causes excessive hair growth in almost all patients.

Related Community Posts Join

6 / 1000+ results

community 0.5mg Dutasteride promotes more “regrowth” than 5mg oral Minoxidil

in Research/Science 117 upvotes 10 months ago

Dutasteride promotes more hair regrowth than Minoxidil. Users discuss combining treatments for better results and share personal experiences with side effects.

community Oral minoxidil is not good for your health, let’s use research chemicals bought from AliExpress

in Satire 551 upvotes 1 year ago

. This conversation discussed the efficacy of Minoxidil and Finasteride as treatments for hair loss, with a focus on the potential side effects of taking Oral Minoxidil. Some users shared their experiences with both treatments and there was debate over whether they were safe or not.

community Androgenetic alopecia is a skin disease: DHT-mediated skin disorders

in Research/Science 62 upvotes 1 year ago

Dihydrotestosterone (DHT) impacts various skin conditions, including Androgenetic alopecia and seborrheic dermatitis, by causing overactivity in sebaceous glands. Topical medications Tacrolimus and Clobetasol can reduce these inflammatory conditions, and treatments like RU58841, Minoxidil, and Finasteride may also be beneficial.

community The Real Cause Of Androgenetic Alopecia

in Research/Science 668 upvotes 2 months ago

Androgenetic alopecia is caused by DHT affecting hair growth. Finasteride and minoxidil are used to manage hair loss by blocking DHT and promoting hair growth.

community My Theory Regarding a Cause and Cure for Androgenetic Alopecia

in Treatment 97 upvotes 4 years ago

Hair loss theory suggests imbalance between Vitamin D Receptor (VDR) and Androgen Receptor (AR) activation. Proposed treatment includes upregulating VDR, downregulating AR, and improving mitochondrial health.

community UPDATE: The theory that explains everything. Please help me make this big!

in Research/Science 441 upvotes 3 years ago

Hair loss theory involves 3alpha-hydroxysteroid reductase (3AHD) converting DHT to androstenol. Discussion explores potential treatments and encourages more research.

Related Research

1 / 1 results

research In Vitro Suppression of Human Lymphocyte Activity by Minoxidil

17 citations, January 1985 in “International Journal of Dermatology”

Minoxidil may help hair growth by stopping immune system attacks on hair follicles.