Laminin 332 Is Indispensable for Homeostatic Epidermal Differentiation Programs
Laminin 332 epidermal homeostasis keratinocyte differentiation epidermal thickening blister formation keratins cellular stress markers actin cytoskeleton inflammatory response S100A8 S100A9 alarmins Lm332 skin homeostasis skin cell differentiation skin thickening skin blisters skin proteins stress markers cell structure inflammation
Studysummary This study found that disrupting Lm332 expression in mice changes keratinocyte genetic expression, alters cell shape, and disrupts epidermal homeostasis, despite some compensatory anchorage by hair follicle basal cells.
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