Expression, Characterization, and Structural Analysis of Human Liver Delta4-3-Ketosteroid 5beta-Reductase (AKR1D1) and Its Disease-Related Mutant P133R

    Jason E. Drury
    TLDR The P133R mutation in AKR1D1 enzyme causes harmful bile acid buildup, explaining related health issues.
    The study focused on the human liver enzyme delta4-3-ketosteroid 5beta-reductase (AKR1D1) and its disease-related mutant P133R. Researchers successfully expressed and purified active recombinant AKR1D1, allowing for detailed structural and functional analysis. The enzyme was capable of 5β-reduction of various steroid precursors, indicating its broad substrate specificity. Structural studies provided the first X-ray crystal structures of AKR1D1 in complex with NADP+ and several steroids, revealing insights into its catalytic mechanism. The P133R mutant exhibited altered kinetic parameters, leading to the accumulation of hepatotoxic allo-bile acids, explaining the clinical symptoms associated with this mutation. This study enhanced the understanding of AKR1D1's role in steroid metabolism and its implications in deficiency syndromes.
    Discuss this study in the Community →