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Understanding hair follicle biology and stem cell control could lead to new hair loss treatments.

Steroid sulfatase is important for activating hormones that affect memory, brain function, and certain diseases, and could be a target for treating hormone-related disorders.

The document concludes that oral finasteride and topical minoxidil are effective for genetic hair loss, while other treatments for different types of hair loss show promise but need more research.

Finasteride effectively lowers specific hormone levels, helping monitor treatment progress.

Drug metabolism affects how long a drug works, its interactions, activation, and toxicity, and is influenced by genetics, diet, illness, and other drugs.

The girl's unexpected pubic hair growth led to a diagnosis different from complete androgen insensitivity syndrome.

Using stem cells from hair follicles to treat female hair loss is safe and effective after six months.

Minoxidil and finasteride are the best for non-scarring hair loss; more research is needed for scarring hair loss treatments.

Combination therapies for androgenetic alopecia work best but can have significant side effects and costs.

Lower levels of certain brain chemicals are linked to worse PTSD symptoms in men.

Minoxidil is processed in the body by an enzyme found in the liver and platelets.

The gel made of minoxidil and hydroxypropyl-β-cyclodextrin improves hair growth and is good for long-term use.

Diazoxide, minoxidil sulphate, and cromakalim relax rat blood vessels by opening K+ channels, with some differences in their actions.

TGF-β2 plays a key role in human hair growth and development.

Minoxidil affects cell growth in two ways: low doses increase growth, while high doses slow it down and can be toxic.

Human platelets change minoxidil to minoxidil sulfate, helping blood vessels widen.

Sulfotransferase in hair follicles helps predict how well minoxidil works for female hair loss.

Minoxidil activation by hair enzymes predicts treatment success for female hair loss.

About 41% of Indian hair loss patients have low enzyme activity that affects hair loss treatment effectiveness, with men affected more than women. Testing for this can guide treatment.

Hair enzyme activity predicts minoxidil success in Brazilian women with hair loss.

Topical minoxidil does not change the activity of hair follicle enzymes that metabolize it.

Minoxidil is less effective on eyebrows and eyelashes than scalp hair because these areas have lower enzyme activity needed to activate the drug.

Minoxidil activates hair growth by being sulfated by P-PST in the human liver.

Liver enzyme helps minoxidil work better for blood vessel relaxation.

Minoxidil works in liver and outer hair root sheath for hair growth.

Scientists identified three genes important for processing certain brain chemicals, thyroid hormones, and medications.

Tretinoin boosts minoxidil's effect on hair loss by increasing enzyme activity.

Topical booster improves hair loss treatment effectiveness.

Some plants like spinach, broccoli, and matcha may boost the effectiveness of the hair growth drug minoxidil.

Low SULT activity in hair follicles leads to better response to oral minoxidil for hair loss.