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Nonclassic congenital adrenal hyperplasia is a genetic disorder causing hormone imbalances, affecting fertility and requiring personalized treatment.

The conclusion is that while oral contraceptive pills are effective for PCOS-related high androgen levels, new treatments with fewer side effects are needed.

The protein StAR is found in 17 different organs and can affect hair loss and brain functions, but its full role is not yet fully understood.

Four new cases of gynecomastia (male breast enlargement) linked to finasteride (Propecia) have been found.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

Certain gene variants may contribute to high androgen levels in women with polycystic ovary syndrome.

Hormone imbalances from the pituitary, thyroid, and adrenal glands can cause infertility, but treating these disorders can improve fertility.

Finasteride treatment changes brain steroid levels and receptors, affecting brain function even after stopping treatment.

More research is needed to understand how testosterone is maintained in adult males.

Triptorelin helps evaluate hormone production in Sertoli-Leydig cell tumors.

Metformin helps with menstrual cycles and insulin levels in PCOS but is less effective for hair growth, diabetes prevention, and weight loss, and may improve fertility and reduce diabetes risk.

Curcumin may improve blood sugar levels and cholesterol in people with PCOS, but more research is needed.

Chemicals and body size might change when puberty starts and progresses, but more research is needed to confirm this.

Modulating Cytochrome P450 activity could help develop new skin disease treatments.

Insulin resistance is linked to PCOS and can lead to other health issues, but treatments like metformin can help manage symptoms.

Congenital Adrenal Hyperplasia is a genetic disorder with two forms, causing symptoms like early puberty and severe acne, but can be identified through screening and treated with glucocorticoids.

Polycystic Ovary Syndrome likely starts in childhood and may be genetic and influenced by early hormone exposure.

The N363S gene variant does not cause higher adrenal androgen levels in women with polycystic ovary syndrome.

The document concludes that more research is needed to fully understand the causes of PCOS.

Finasteride may improve prostate cancer treatment outcomes.

Young girls whose mothers have PCOS may have higher activity of a specific enzyme that could lead to developing PCOS later.

Skin lesions in Carney complex are likely caused by a specific group of skin cells that promote pigment production due to a genetic mutation.

Human sebaceous glands contain enzymes that affect androgen production and may influence sebum production and acne.

Gut microbes significantly affect brain steroid levels.

The research found that polycystic ovary syndrome (PCOS) has two distinct types, with one having more severe hormone and insulin issues.

Chromosomal differences affect how muscle cells respond to testosterone.

Oestrogens help maintain healthy skin, heal wounds, and may protect against skin aging and cancer.

Most adrenal cortex tumors are benign and non-secreting, but proper diagnosis and treatment are important due to the rare possibility of cancer.

Hormones and signaling pathways control sebaceous gland function and could help treat acne.

Lipin-1 is important for skin cell differentiation and skin barrier function.