Search
for
Sort by
Research 31–60 of 48
- Targeting the Hippo/YAP/TAZ signalling pathway: Novel opportunities for therapeutic interventions into skin cancers
- Volume imaging to interrogate cancer cell-tumor microenvironment interactions in space and time
- Imaging to study solid tumour origin and progression: lessons from research and clinical oncology
- Extracellular Vesicles in Regenerative and Cosmetic Medicine: Safety, Clinical Effectiveness, Therapeutic Applications, and Regulatory Challenges
- Image2.TIF
- DataSheet1.zip
- Image1.TIF
- Presentation1.PPTX
- Development and homeostasis of the sebaceous gland
- PROTACs: A novel strategy for cancer drug discovery and development
- Current and prospective strategies for advancing the targeted delivery of CRISPR/Cas system via extracellular vesicles
- Acute sensitivity of the oral mucosa to oncogenic <i>K‐ras</i>
- Advancing Design Strategy of PROTACs for Cancer Therapy
- Proteolysis‐targeting chimeras in cancer therapy: Targeted protein degradation for next‐generation treatment
- Transcriptome-guided development of a fibrosis-reversal compound reduces skin scarring and allows regeneration via mitochondrial uncoupling
- BAYONET trial: staged combination with encorafenib, binimetinib, plus cetuximab following encorafenib plus cetuximab for BRAF V600E-mutant metastatic colorectal cancer
- New frontiers in CRISPR/Cas9 delivery systems delivery for gene editing
- Montagna Symposium 2015: Harnessing Stem Cells to Reveal Novel Skin Biology and Disease Treatments
- KRASG12D mutant cells are outcompeted by wild type neighbours in adult pancreas in an EPHA2-dependent manner
- YAP and TAZ are essential for basal and squamous cell carcinoma initiation
- Stem cell quiescence acts as a tumour suppressor in squamous tumours
- Activated Kras Alters Epidermal Homeostasis of Mouse Skin, Resulting in Redundant Skin and Defective Hair Cycling
- Tumor suppressor identity can contribute to heterogeneity of phenotype in hair follicle stem cell‐induced squamous cell carcinoma
- LB1011 Oncogenic ras mutation induces spatiotemporally specific tissue deformation through converting fluctuated into sustained ERK activation
- Oncogenic Kras induces spatiotemporally specific tissue deformation through converting pulsatile into sustained ERK activation
- Injury suppresses Ras cell competitive advantage through enhanced wild-type cell proliferation
- Drug Repurposing against KRAS Mutant G12C: A Machine Learning, Molecular Docking, and Molecular Dynamics Study
- Tracing the cellular origin of cancer
- Extracellular vesicle-based drug overview: research landscape, quality control and nonclinical evaluation strategies
- Roles of tumor suppressors in regulating tumor-associated inflammation