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Hair quality is genetically determined and linked to its composition and strength.

Psoriasis is a common, genetically influenced skin disease worsened by stress and lifestyle, but targeted treatments are promising.

Acne is genetically linked to a higher risk of depression, anxiety, schizophrenia, and bipolar disorder, with the strongest link to bipolar disorder.

Vitamin D3 analogs can promote hair growth in mice genetically prone to hair loss.

COVID-19 vaccines may cause hair loss in people who are genetically prone to it.

COVID-19 can lead to different skin symptoms and might trigger autoimmune diseases in genetically susceptible people.

A woman had both Graham Little-Piccardi-Lassueur syndrome, causing hair loss, and complete androgen insensitivity syndrome, making her genetically male but physically female. This suggests androgens don't affect the hair loss condition.

Male pattern baldness is an unintended side effect of the body's use of androgens for muscle growth, especially in those genetically prone to it.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

Skin grafting and wound treatment have improved, but we need more research to better understand wound healing and create more effective treatments.

Scientists turned mouse stem cells into skin cells that can grow into skin layers and structures.

FPHL is a common, age-related, genetic hair loss with unclear causes and limited treatment options.

Japanese vitiligo patients and their families often have other autoimmune diseases.

Hair grays due to oxidative stress and fewer functioning melanocytes.

Hair thinning causes stem cell loss through a process involving Piezo1, calcium, and TNF-α.

Women with low SHBG levels and a high 3a-diol G to SHBG ratio are likely to experience female pattern baldness, possibly due to a slight excess of androgens affecting sensitive hair bulbs.

Explosions don't stop hair proteins from being used to identify people.

The best animal model for studying male-pattern baldness is the stumptailed macaque, not rats or mice.

Two siblings both had a rare case of alopecia areata at the same time.

The CUBIC protocol allows detailed 3D visualization of proteins in mouse skin biopsies.

The research showed how melanocytes develop, move, and respond to UV light, and their stem cells' role in hair color and skin cancer risk.

Emerging therapies like stem cell and laser treatments show promise for hair regeneration.

Frontal Fibrosing Alopecia is a hair loss condition mainly affecting postmenopausal women, with unclear causes and various clinical patterns.

The document concludes that identifying the specific cells where skin cancers begin is important for creating better prevention, detection, and treatment methods.

Forensic DNA phenotyping is becoming useful for predicting physical traits in criminal investigations but is limited by ethical concerns and incomplete genetic understanding.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

The mutation helps mice handle heat better without affecting hair growth.

Testosterone significantly affects urination differences between male and female mice.

The conclusion is that having both Frontal Fibrosing Alopecia and Discoid Lupus Erythematosus may suggest a shared immune response in certain people, and a mix of antimalarial drugs and 5-alfa-reductase inhibitors is recommended for treatment.

Changes in skin bacteria can affect hair loss and new treatments targeting these bacteria may prevent balding without sexual side effects.