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Children with metabolic syndrome often have skin problems like dark patches, skin tags, stretch marks, infections, acne, and psoriasis, which are linked to obesity and insulin resistance.

The document concludes that Syndromes of Severe Insulin Resistance are rare disorders with limited treatment options.

Women with PCOS have similar levels of certain proteins compared to women without PCOS, and these proteins don't independently cause PCOS.

Women with PCOS often have more belly fat and higher insulin levels, but belly fat isn't the only cause of their insulin resistance.

Women with PCOS have overall higher body fat, not abnormal fat placement, and more insulin resistance.

Both immature and mature fat cells are important for hair growth cycles, with immature cells promoting growth and mature cells possibly inhibiting it.

Women with PCOS have more body fat and thicker fat layers in certain abdominal areas than women without PCOS.

Daughters of women with PCOS generally experience normal puberty but may have more body hair and different body fat distribution.

Gender-affirming hormone therapy improves physical performance in trans men to the level of cisgender men, while in trans women, it increases fat mass and decreases muscle mass, with no advantage in physical performance after 2 years.

Obese women with PCOS show a male-like pattern in certain fat tissue gene expressions.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Treating sleep apnea in young obese women with PCOS can improve heart health and insulin sensitivity.

Skin fat has important roles in hair growth, skin repair, immune defense, and aging, and could be targeted for skin and hair treatments.

Human hair follicles can make and process prostaglandins, which may affect hair growth.

Glutamine metabolism affects hair stem cell maintenance and their ability to change back to stem cells.

Hair loss in male pattern baldness involves muscle degeneration and increased scalp fat.

More PDCD4 is linked to obesity, insulin problems, and cell death in ovaries for those with polycystic ovary syndrome.

Benign Prostatic Hyperplasia may be caused by changes in how the body processes male hormones.

Altered retinoid metabolism in cicatricial alopecia suggests a balanced vitamin A diet may prevent the condition.

Androgens affect bone and fat cell development differently based on the cells' embryonic origin.

Testosterone conversion to 5α-DHT may not be essential for its effects on the skin.

People with hidradenitis suppurativa often have lower vitamin D levels, weaker hip bones, and altered bone metabolism markers.

Taking a probiotic supplement twice a day improved hair growth, skin hydration, and metabolic health in adults at risk of metabolic syndrome.

Testosterone therapy improves physical function, strength, and body composition in older men with low testosterone levels.

Some wound-healing cells can turn into fat cells around new hair growth in mice.

The testosterone patch and gel are both effective, but they have different absorption patterns and effects on hormone levels.

Both mesotherapy treatments reduced under-chin fat equally and were safe with mild side effects.

Lepidium sativum seed extracts helped reduce inflammation and improve insulin response in obese rats on a high-fat diet.

Children with classic congenital adrenal hyperplasia have thicker heart fat and are at higher risk for heart problems and early atherosclerosis.

Rat skin takes up and processes testosterone differently than other organs, with testosterone being more important for the skin than its metabolite 5α-DHT.