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Vav2 changes how hair follicle stem cells' genes work as they age, which might improve regeneration but also raise cancer risk.

A group has developed therapies that show promise for treating cancer and various other conditions.

Water and fatty acids affect hair's surface differently based on hair damage, and models can help understand hair-cosmetic interactions.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

PlacMA hydrogels from human placenta are versatile and useful for cell culture and tissue engineering.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

Human hair follicle cells can be turned into stem cells that may help clone hair for treating hair loss or burns.

HNG may help prevent the negative effects of chemotherapy on sperm production and white blood cell counts.

Hair follicles can be used to quickly assess drug effects in cancer treatment.

TRPV3 could be a target for treating pain, skin disorders, and hair problems, but more research is needed to create effective drugs.

The document concludes that managing skin conditions during pregnancy is important and requires specialized care.

Two microRNAs affect hair follicle development in sheep by targeting specific genes.

Hyaluronic acid and polycaprolactone improve skin regeneration, with polycaprolactone having a stronger effect on healing and tissue repair.

The SbbHLH85 protein helps sweet sorghum grow more root hairs but makes the plant more sensitive to salt.

The document concludes that most hair loss treatments don't work, balding isn't caused by dandruff, and hair loss may indicate serious health issues that require medical attention.

Atopic dermatitis may have genetic causes and can be treated with pharmacologic methods, glycerin creams, and controlling Staphylococcus aureus colonization.

The calcineurin/NFAT pathway plays a significant role in the development and growth of a type of skin cancer called cutaneous squamous cell carcinoma.

Androgen self-administration might be controlled by membrane receptors, not nuclear ones.

Minoxidil may work for hair loss by reducing androgen sensitivity and altering hormone-related enzymes.

Small proline-rich proteins and trichohyalin help make epithelial tissues tougher and more flexible.

Different signals work together to change gene activity and guide hair follicle stem cells to become specific cell types.

Deleting MED1 in skin cells causes hair loss and skin changes.

The vitamin D receptor can work without its usual activating molecule.

The lentiviral array can monitor and predict gene activity during stem cell differentiation.

The HPV type 11 region activates hair-specific gene expression in mice.

Different ligands change the shape of the TRPV3 ion channel in unique ways.

Using minoxidil-coated microbubbles with ultrasound significantly boosts hair growth.

Certain skin proteins can form anchoring structures without the protein AMACO.

Enzymes are classified into six types and are essential for many biological processes, with only a few targeted by drugs.