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Research 30 of 506
- Overexpression of activin A in the skin of transgenic mice reveals new activities of activin in epidermal morphogenesis, dermal fibrosis and wound repair
- Activin Controls Skin Morphogenesis and Wound Repair Predominantly via Stromal Cells and in a Concentration-Dependent Manner via Keratinocytes
- The roles of activins in repair processes of the skin and the brain
- Conditional Activin Receptor Type 1B (Acvr1b) Knockout Mice Reveal Hair Loss Abnormality
- The activin binding proteins follistatin and follistatin-related protein are differentially regulated in vitro and during cutaneous wound repair
- The role of activins and follistatins in skin and hair follicle development and function
- GLI2-specific Transcriptional Activation of the Bone Morphogenetic Protein/Activin Antagonist Follistatin in Human Epidermal Cells
- Regulatory T cells are required for normal and activin‐promoted wound repair in mice
- A counter gradient of Activin A and follistatin instructs the timing of hair cell differentiation in the murine cochlea
- Activin A-induced signalling controls hair follicle neogenesis
- Faculty Opinions recommendation of A counter gradient of Activin A and follistatin instructs the timing of hair cell differentiation in the murine cochlea.
- Author response: A counter gradient of Activin A and follistatin instructs the timing of hair cell differentiation in the murine cochlea
- Decision letter: A counter gradient of Activin A and follistatin instructs the timing of hair cell differentiation in the murine cochlea
- Activin A and Follistatin influence hair follicle development in mice
- Impaired wound healing in transgenic mice overexpressing the activin antagonist follistatin in the epidermis
- Expression and Function of Keratinocyte Growth Factor and Activin in Skin Morphogenesis and Cutaneous Wound Repair
- Control of pelage hair follicle development and cycling by complex interactions between follistatin and activin
- Activin B Promotes BMSC-Mediated Cutaneous Wound Healing by Regulating Cell Migration via the JNK—ERK Signaling Pathway
- Activin B Promotes Epithelial Wound Healing In Vivo through RhoA-JNK Signaling Pathway
- Activin B Stimulates Mouse Vibrissae Growth and Regulates Cell Proliferation and Cell Cycle Progression of Hair Matrix Cells through ERK Signaling
- Activin B Promotes Initiation and Development of Hair Follicles in Mice
- Supplementary Material for: Activin B Promotes Initiation and Development of Hair Follicles in Mice
- Activin/Nodal signalling in stem cells
- Expression of <i>Foxi3</i> is regulated by ectodysplasin in skin appendage placodes
- Single-Cell RNA Profiling of Human Skin Reveals Age-Related Loss of Dermal Sheath Cells and Their Contribution to a Juvenile Phenotype
- Isoproterenol directs hair follicle-associated pluripotent (HAP) stem cells to differentiate <i>in vitro</i> to cardiac muscle cells which can be induced to form beating heart-muscle tissue sheets
- Impact of One-Week Administration of Dihydrotestosterone in Rat Anterior Pituitary Gland
- Rat hair-follicle-associated pluripotent (HAP) stem cells can differentiate into atrial or ventricular cardiomyocytes in culture controlled by specific supplementation
- Physiology of Male Gonadotropic Axis and Disorders of Sex Development
- Overexpression of Smad7 results in severe pathological alterations in multiple epithelial tissues