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GlossaryMICA

protein involved in immune response and stress detection

MICA stands for MHC class I polypeptide-related sequence A. It is a protein that is part of the immune system and is involved in the body's response to stress and infection. MICA is expressed on the surface of certain cells and can be recognized by natural killer (NK) cells, which play a role in targeting and destroying infected or cancerous cells. In the context of hair and alopecia, variations in the MICA gene have been studied for their potential association with autoimmune conditions like alopecia areata.

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research Joint Study of the Associations of HLA-B and the Transmembrane Short Tandem Repeat Polymorphism of MICA Protein with Alopecia Areata Shows Independent Associations of Both with the Disease

7 citations, February 2020 in “Clinical and Experimental Dermatology”

Both HLA-B and MICA are independently linked to alopecia areata.

research Alopecia Areata Is Associated With MICA and an Extended HLA Haplotype

1 citations, March 2004 in “Journal of the American Academy of Dermatology”

Certain genes are linked to the risk of developing Alopecia Areata.

research Environmental Pathobiology of Frontal Fibrosing Alopecia: Does Linalool Promote FFA Development and Progression?

November 2022 in “The journal of investigative dermatology/Journal of investigative dermatology”

Linalool in personal care products may contribute to hair loss by damaging hair follicle stem cells and triggering harmful immune responses.

research Targeting Pathogenic MICA-NKG2D Interactions by Statins: A Novel Adjunct Treatment Strategy for Alopecia Areata Management?

November 2022 in “Journal of Investigative Dermatology”

Statins may help treat alopecia areata by reducing harmful immune interactions.

research Environmental Pathobiology of Frontal Fibrosing Alopecia: Does Linalool Promote FFA Development and Progression by Inducing Epithelial Hair Follicle Stem Cell Damage and MICA Expression in the Bulge?

September 2022

Linalool in personal care products may worsen frontal fibrosing alopecia by damaging hair follicle stem cells and triggering harmful immune responses.

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