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- Fetal Programming of Polycystic Ovary Syndrome by Androgen Excess: Evidence from Experimental, Clinical, and Genetic Association Studies
- FETAL METABOLIC PROGRAMMING IN THE ETIOLOGY OF POLYCYSTIC OVARIAN SYNDROME
- Rodent models in placental research. Implications for fetal origins of adult disease
- Timing of puberty in relation to semen characteristics, testicular volume, and reproductive hormones: a cohort study
- Premature adrenarche, polycystic ovary syndrome and intrauterine growth retardation: does a relationship exist?
- Tissue resident and follicular Treg cell differentiation is regulated by CRAC channels
- Polycystic ovary syndrome and impact on health
- Maternal androgen excess inhibits fetal cardiomyocytes proliferation through RB-mediated cell cycle arrest and induces cardiac hypertrophy in adulthood
- Contaminants of emerging concern in the fetal environment
- Animal Models and the Developmental Origins of Polycystic Ovary Syndrome: Increasing Evidence for the Role of Androgens in Programming Reproductive and Metabolic Dysfunction
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