Search
for

    Glossary Bcl-2 family

    regulates cell death and survival through protein interactions

    The Bcl-2 family, also known as the B-cell lymphoma 2 family, is a group of proteins that play a crucial role in regulating cell death (apoptosis). These proteins can either promote survival or trigger cell death, and their balance is essential for maintaining healthy tissue function and preventing diseases like cancer.

    Related Terms

    Research 10 of 1000+

    1. Protein kinase Akt2/PKBβ is involved in blastomere proliferation of preimplantation mouse embryos Journal of Cellular Physiology · 2019 · 7 citations
    2. Цинк-зависимые факторы в регуляции цикла фолликула волоса Международный журнал прикладных и фундаментальных исследований · 2014
    3. Synthesized Ceramide Induces Growth of Dermal Papilla Cells with Potential Contribution to Hair Growth 2019 · 7 citations
    4. Alex de la Iglesia: "Todos somos perversos" Cambio 16 · 2000
    5. ARTS, an unusual septin, regulates tumorigenesis by promoting apoptosis Biomedicine & Pharmacotherapy · 2022 · 10 citations
    6. Expression of COX-2 and Bcl-2 in 50 patients of Lichen Planus International journal of clinical and diagnostic pathology · 2020
    7. Hydroxyl radical mediates cisplatin-induced apoptosis in human hair follicle dermal papilla cells and keratinocytes through Bcl-2-dependent mechanism APOPTOSIS · 2011 · 52 citations
    8. Overexpression of Bcl-2 Protects from Ultraviolet B-Induced Apoptosis but Promotes Hair Follicle Regression and Chemotherapy-Induced Alopecia ˜The œAmerican journal of pathology · 2000 · 47 citations
    9. A medicinal and edible formula YH0618 ameliorates the toxicity induced by Doxorubicin via regulating the expression of Bax/Bcl-2 and FOXO4 Journal of cancer · 2019 · 17 citations
    10. The anti‐apoptotic Bcl‐2 protein regulates hair follicle stem cell function EMBO Reports · 2021 · 10 citations
    All research results →

    Learn

    — no results

    Try a deeper search in learn →

    Community

    — no results

    Try a deeper search in community →