Tyrosinase Inhibition Due to Interaction of Homocysteine with Copper: Mechanism for Reversible Hypopigmentation in Homocystinuria Due to Cystathionine Beta-Synthase Deficiency

    July 1995 in “ PubMed ”
    Orit Reish, Danyelle M. Townsend, Susan A. Berry … Richard A. King
    Studysummary This study suggests that reversible hypopigmentation in homocystinuric patients may result from tyrosinase inhibition by homocyst(e)ine, with in-vitro experiments showing copper sulfate reversing this inhibition.
    Automatically generated from the study's abstract, not written by a person, and not a review of the full paper. Not medical advice or a treatment recommendation. Read the original study, and consult a qualified healthcare professional before changing treatment. Full disclaimer
    Read the full study on pubmed.ncbi.nlm.nih.gov →
    Discuss this study in the Community →

    Related research 1

    1. Tyrosinase Inhibition Due to Interaction of Homocysteine with Copper: Mechanism for Reversible Hypopigmentation in Homocystinuria Due to Cystathionine Beta-Synthase Deficiency PubMed · 1995