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- Towards Defining the Pathogenesis of the Hairless Phenotype
- Endogenous Skin Fluorescence is a Good Marker for Objective Evaluation of Comedolysis
- Structural changes in hair follicles and sebaceous glands of hairless mice following exposure to sulfur mustard
- Relation of Skin Polyamines to the Hairless Phenotype in Transgenic Mice Overexpressing Spermidine/Spermine N1-Acetyltransferase
- Disturbed Keratinocyte Differentiation in Transgenic Mice and Organotypic Keratinocyte Cultures as a Result of Spermidine/Spermine N1-Acetyltransferase Overexpression
- Mesenchymal stem cell-derived exosomes ameliorate gentamicin-induced vestibular hair cell injury by regulating the SNARE pathway and enhancing autophagy
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