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- Skin abnormalities generated by temporally controlled RXRα mutations in mouse epidermis
- [Genetic dissection of retinoic acid function in epidermis physiology].
- Functional analyses of a novel missense and other mutations of the vitamin D receptor in association with alopecia
- Physiological and retinoid-induced proliferations of epidermis basal keratinocytes are differently controlled
- Disruption of the hedgehog signaling pathway contributes to the hair follicle cycling deficiency in Vdr knockout mice
- Hairless is a nuclear receptor corepressor essential for skin function
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- Mediator1: An Important Intermediary of Vitamin D Receptor–Regulated Epidermal Function and Hair Follicle Biology
- Ligand‐Independent Actions of the Vitamin D Receptor: More Questions Than Answers
- Genetic Disorders and Defects in Vitamin D Action
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