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Research 30 of 601
- THY1-mediated mechanisms converge to drive YAP activation in skin homeostasis and repair
- 277 HPV8 E6 induced STAT3 activation leads to hair follicle junctional zone keratinocyte stem cell proliferation and expansion in actinic keratoses
- Langerin+ Dendritic Cells in Cutaneous Fibrosis: The TGF-β1 Signaling Axis
- Caspase-3 Regulates YAP-Dependent Cell Proliferation and Organ Size
- Many Paths to Alopecia via Compromised Regeneration of Hair Follicle Stem Cells
- Cold atmospheric microwave plasma (CAMP) stimulates dermal papilla cell proliferation by inducing β-catenin signaling
- YAP1 promotes adipogenesis by regulating the negative feedback mechanism of the Hippo signaling pathway via LATS2
- CuATSM Enhances Wound Repair Without Scarring via Hippo/YAP Signalling Pathway to Reduce Ferroptosis and Macrophage Polarisation
- Epithelial Fibronectin meshwork controls skin regeneration
- Novel Tissue Mechanics-Guided Cellular Flows Enable the Evolution of Feather Follicles
- MicroRNA-205 promotes hair regeneration by modulating mechanical properties of hair follicle stem cells
- Colorectal cancer co-opts an epidermal wound healing program during metastasis to generate disseminated tumor cells
- Canonical and emerging regulatory mechanisms of tissue remodeling: shared principles across organs and therapeutic opportunities
- A Consensus Single-cell Atlas Of Hidradenitis Suppurativa Tunnels Defines A Fibro-inflammatory Niche With Druggable Microenvironmental Endotypes
- A Consensus Single-cell Atlas Of Hidradenitis Suppurativa Tunnels Defines A Fibro-inflammatory Niche With Druggable Microenvironmental Endotypes
- A Consensus Single-cell Atlas Of Hidradenitis Suppurativa Tunnels Defines A Fibro-inflammatory Niche With Druggable Microenvironmental Endotypes
- A Consensus Single-cell Atlas Of Hidradenitis Suppurativa Tunnels Defines A Fibro-inflammatory Niche With Druggable Microenvironmental Endotypes
- YAP and TAZ are essential for basal and squamous cell carcinoma initiation
- EDA Fibronectin Microarchitecture and YAP Translocation During Wound Closure
- Mechanical stretch induces hair regeneration through the alternative activation of macrophages
- Metabolic and transcriptomic reprogramming during contact inhibition-induced quiescence is mediated by YAP-dependent and YAP-independent mechanisms
- Role of β-Catenin Activation Levels and Fluctuations in Controlling Cell Fate
- Targeting the Hippo/YAP/TAZ signalling pathway: Novel opportunities for therapeutic interventions into skin cancers
- Integrin signalling regulates YAP/TAZ to control skin homeostasis
- The therapeutic promise of apoptosis
- YAP/TAZ Signalling Controls Epidermal Keratinocyte Fate
- Molecular pathology of skin adnexal tumours
- Cthrc1 deficiency aggravates wound healing and promotes cardiac rupture after myocardial infarction via non-canonical WNT5A signaling pathway
- Radiotherapy-Induced Skin Fibrosis: Pathophysiology, Emerging Therapeutics, and the Role of Dermatology
- Hair regeneration: Mechano-activation and related therapeutic approaches.