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PRP and PRF can effectively heal chronic wounds.

Mice lacking fibromodulin have disrupted healing patterns, leading to abnormal skin repair and scarring.

The fascial layer is a promising new target for wound healing treatments using biomaterials.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Hair follicle bulge cells are important for hair survival and help heal the skin after injury, which might be relevant for understanding hidradenitis suppurativa.

M-CSF-stimulated myeloid cells can turn into skin cells and help heal wounds and regrow hair.

The research found that different types of fibroblasts are involved in wound healing and that some blood cells can turn into fat cells during this process.

Stem cells are crucial for wound healing and understanding their role could lead to new treatments, but more research is needed to answer unresolved questions.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Epidermal stem cells help heal severe skin wounds and have potential for medical treatments.

Fibroblast state switching is crucial for skin healing and development.

Scarless healing is complex and influenced by genetics and environment, while better understanding could improve scar treatment.

Skin bacteria, specifically Staphylococcus aureus, help in wound healing and hair growth by using IL-1β signaling. Using antibiotics on skin wounds can slow down this natural healing process.

Modified rat stem cells on a special scaffold improved blood vessel formation and wound healing in skin substitutes.

Activin B helps heal skin wounds and grow hair by activating a specific cell signaling pathway.

Fibromodulin treatment helps reduce scarring and improves wound healing by making it more like fetal healing.

Vitamin D receptor is important for regulating hair growth and wound healing in mice.

Inflammation plays a key role in activating skin stem cells for hair growth and wound healing, but more research is needed to understand how it directs cell behavior.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

The biofilm-dispersing wound gel helps wounds heal faster and prevents infection.

Gene therapy shows promise for improving wound healing, but more research is needed for human use.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

Thy-1 protein helps improve blood flow and wound healing in the skin.

Regulatory T cells help heal skin and grow hair, and their absence can lead to healing issues and hair loss.

A topical BRAF inhibitor, vemurafenib, can speed up wound healing and promote hair growth, especially in diabetic patients.

Fat-derived stem cells show promise for treating skin issues and improving wound healing, but more research is needed to confirm the best way to use them.

Nanofiber scaffolds help wounds heal by delivering drugs directly to the injury site.