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Activin A promotes ear hair cell development, while follistatin delays it.

The Notch signaling pathway is important for hair follicle development and could help create treatments for hair disorders.

The 2015 Hair Research Congress concluded that stem cells, maraviroc, and simvastatin could potentially treat Alopecia Areata, topical minoxidil, finasteride, and steroids could treat Frontal Fibrosing Alopecia, and PTGDR2 antagonists could also treat alopecia. They also found that low-level light therapy could help with hair loss, a robotic device could assist in hair extraction, and nutrition could aid hair growth. They suggested that Alopecia Areata is an inflammatory disorder, not a single disease, indicating a need for personalized treatments.

ALRV5XR is the most effective hair regrowth treatment at 24 weeks.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

Tcf3 helps cells move and heal wounds by controlling lipocalin 2.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

Hair follicles can regrow in wounded adult mouse skin using a process like embryo development.

Wnt signaling is important for development and cell regulation but can cause diseases like cancer when not working properly.

Androgen receptor activity blocks Wnt/β-catenin signaling, affecting hair growth and skin cell balance.

Mouse cell exosomes help hair regrowth and wound healing by activating a specific signaling pathway.

Different types of stem cells and their environments are key to skin repair and maintenance.

The conclusion is that tissue structure is key for stem cell communication and maintaining healthy tissues.

R-spondins and their receptors help increase bone growth and may be used to treat bone loss diseases.

Removing the VDR gene in skin cells reduces their growth and affects hair-related genes.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

Vitamin D receptor may help protect against UV-induced skin cancer.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Hair, teeth, and mammary glands develop similarly at first but use different genes later.

Activating β-catenin in different skin stem cells causes various types of hair growth and skin tumors.

Super-enhancers controlled by pioneer factors like SOX9 are crucial for stem cell adaptability and identity.

Hair follicle dermal stem cells are key for regenerating parts of the hair follicle and determining hair type.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Vitamin D has potential benefits for cancer prevention, heart health, diabetes, obesity, muscle function, skin health, and immune function, but clinical results are mixed and more research is needed.

The extracellular matrix is crucial for controlling skin stem cell behavior and health.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

Hormones and neuroendocrine factors control hair growth and color, and more research could lead to new hair treatment options.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

Vitamin D and calcium are important for quick and effective skin wound healing.