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WNT10A mutations cause varied symptoms in patients with odonto-onycho-dermal dysplasia.

GasderminA3 is important for normal hair cycle transitions by controlling Wnt signaling.

Rats can't grow new hair follicles after skin wounds, unlike mice, due to differences in gene expression and response to WNT signaling.

Prunus mira nut oil helps hair grow by affecting the Wnt/β-catenin signaling pathway.

Researchers found WNT10A to be a key gene in developing goat hair follicles.

Oleanolic acid promotes hair growth by increasing cell proliferation through the Wnt/β-catenin pathway.

Nail stem cells and Wnt signaling are important for fingertip regeneration but not sufficient for regenerating more complex limb structures.

Wounds can cause new hair growth in adult mice, influenced by Wnt signaling.

LEF1 is essential for the development of airway glands and is regulated by the Wnt/ß-catenin pathway.

WNT7A gene expression is higher in early stages of androgenetic alopecia, showing the role of WNT pathway, apoptosis, and inflammation in the disorder.

New research shows that skin diversity is influenced by different types of dermal fibroblasts and their development, especially involving the Wnt/β-catenin pathway.

WNT10A gene mutations cause short anagen hair syndrome.

New treatments for hair loss could involve using stem cells and a process called the Wnt/beta-catenin pathway to stimulate hair growth.

Healing skin wounds in mice can create new hair follicles, and adjusting Wnt signaling could potentially reduce scarring and treat hair loss.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

A new change in the WNT10A gene caused a condition leading to short hair growth in a Chinese family.

Mutations in the WNT10A gene cause Short Anagen Hair syndrome and increase the risk of male pattern hair loss.

Arrector pili muscle regulates hair follicle stem cells, DNA methylation needed for hair cycling, and Wnt/B-catenin signaling starts hair growth.

Skin stem cells are crucial for maintaining and repairing the skin and hair, using a complex mix of signals to do so.

Understanding hair follicle development can help treat hair loss, skin regeneration, and certain skin cancers.

The document concludes that skin stem cells are important for hair growth and wound healing, and could be used in regenerative medicine.

Hair follicles are complex, dynamic mini-organs that help us understand cell growth, death, migration, and differentiation, as well as tissue regeneration and tumor biology.

Epithelial stem cells are crucial for tissue renewal and repair, and understanding them could improve treatments for damage and cancer.

β-catenin is essential for stem cell activation and proliferation in hair follicles.

The document concludes that the hair cycle is a complex process involving growth, regression, and rest phases, regulated by various molecular signals.

The conclusion is that certain cell interactions and signals are crucial for hair growth and regeneration.

Different types of fibroblasts play various roles in diseases and healing, and more research on them could improve treatments.

Stromal stem cells may help heal wounds by becoming structural cells or affecting the immune system, but more research is needed to understand how.

Notch signalling helps skin cells differentiate and prevents tumors.

Ectodin integrates BMP, SHH, and FGF signals in developing ectodermal organs.